1. Complexation. ① Heavy metal poisoning: this product can complex heavy metals such as copper, iron, mercury, lead, and arsenic to form stable and soluble complexes that are excreted in urine. Its lead removal effect is not as good as that of calcium sodium edetate, and its mercury removal effect is not as good as that of dithiocarbamide; but this product can be taken orally, with slightly fewer adverse reactions, and can be used for mild heavy metal poisoning or when other complexing agents are contraindicated. ② Wilson's disease is a common chromosomal recessive genetic disease, mainly with a large amount of copper deposited in the liver and brain tissues, causing lenticular degeneration and cirrhosis. This product can combine with copper deposited in tissues to form soluble complexes that are excreted in urine. ③ Cystinuria and its stones: this product can react with cystine to form a mixture of cysteine-penicillamine disulfide, thereby reducing the concentration of cystine in urine; the solubility of this mixture is 50 times greater than that of cystine, so it can prevent the formation of cystine stones; long-term use for 6 to 12 months may gradually dissolve the already formed cystine stones. 2. Anti-rheumatoid arthritis: The mechanism of action for the treatment of rheumatoid arthritis is not yet clear. After taking the medicine, it was found that the lymphocyte function was improved, and the levels of IgM rheumatoid factor and immune complex in serum and joint capsule fluid were significantly reduced, but there was no significant reduction in the absolute value of serum immunoglobulin. It inhibited the activity of T cells in vitro, but had no effect on B cells. This product can also inhibit the cross-linking of newly synthesized procollagen, so it is also used to treat skin and soft tissue collagen diseases.

No specific pharmacological effects mentioned

Oral medication for the treatment of pregnancy-induced hypertension has the effects of dilating peripheral blood vessels and inhibiting uterine activity. Its main mechanism may be to increase the concentration of Mg++ in the blood and extracellular fluid, thereby directly dilating the peripheral vascular smooth muscle and reducing the tension of uterine smooth muscle.

It is rapidly converted into the antiviral compound penciclovir in the body, which has an inhibitory effect on herpes simplex virus type Ⅰ (HSV-1), herpes simplex virus type Ⅱ (HSV-2) and varicella zoster virus (VZV). It inhibits the activity of HSV-2 polymerase by competing with guanosine triphosphate, thereby selectively inhibiting the synthesis and replication of herpes virus DNA.

Extract from the above information