On ECHEMI
Home > Drugs > Beijing Sihuan Kebao Pharmaceutical Co., Ltd.
Beijing Sihuan Kebao Pharmaceutical Co., Ltd.
  • Founded in:

    1998-12-04
  • Country:

    China China
  • Address:

    No. 11, Haiying Road, Science and Technology Park, Fengtai District, Beijing [Park]
  • Tax NO.:

    91110106700101574K
  • Registered Funds:

    150 million yuan
  • Website:

  • Email:

Related Drugs
Thymosin for injection
Thymosin α1 and other small peptides
Name Description Content CAS NO. Registered Holders
thymosin1, Alpha1

This product is an immunomodulatory drug. It has the function of regulating and enhancing the cellular immune function of the human body and can promote the maturation of T-lymphocytes.

More

This product is an immunomodulatory drug. It has the function of regulating and enhancing the cellular immune function of the human body and can promote the maturation of T-lymphocytes.

0
Pinealon

Pharmacodynamics: This product is an immunomodulatory drug. It has the function of regulating and enhancing the cellular immune function of the human body and can promote the maturation of T-lymphocytes.

More

Pharmacodynamics: This product is an immunomodulatory drug. It has the function of regulating and enhancing the cellular immune function of the human body and can promote the maturation of T-lymphocytes.

0
Isosorbide dinitrate for injection
Isosorbide dinitrate
Name Description Content CAS NO. Registered Holders
Isosorbide dinitrate

It relaxes vascular smooth muscle, releases nitric oxide through metabolism to generate isosorbide mononitrate, activates guanylate cyclase, increases cyclic guanosine monophosphate in smooth muscle cells, and thus relaxes vascular smooth muscle. It dilates peripheral arteries and veins, reduces the amount of blood returning to the heart, reduces left ventricular end-diastolic pressure and pulmonary capillary wedge pressure (preload), reduces peripheral vascular resistance, systolic arterial pressure and mean arterial pressure (afterload), increases coronary artery perfusion, reduces myocardial oxygen consumption and increases oxygen supply.

More

It relaxes vascular smooth muscle, releases nitric oxide through metabolism to generate isosorbide mononitrate, activates guanylate cyclase, increases cyclic guanosine monophosphate in smooth muscle cells, and thus relaxes vascular smooth muscle. It dilates peripheral arteries and veins, reduces the amount of blood returning to the heart, reduces left ventricular end-diastolic pressure and pulmonary capillary wedge pressure (preload), reduces peripheral vascular resistance, systolic arterial pressure and mean arterial pressure (afterload), increases coronary artery perfusion, reduces myocardial oxygen consumption and increases oxygen supply.

87-33-2 23
Isosorbide dinitrate for injection
Isosorbide dinitrate
Name Description Content CAS NO. Registered Holders
Isosorbide dinitrate

It relaxes vascular smooth muscle, releases nitric oxide (NO) through metabolism to produce isosorbide mononitrate (ISMN), activates guanylate cyclase, and increases cyclic guanosine monophosphate (cGMP) in smooth muscle cells, thereby relaxing vascular smooth muscle, dilating peripheral arteries and veins, reducing the amount of blood returning to the heart, lowering left ventricular end-diastolic pressure and pulmonary capillary wedge pressure (preload), reducing peripheral vascular resistance, systolic arterial pressure and mean arterial pressure (afterload), dilating coronary arteries and increasing coronary perfusion. The overall effect is to reduce myocardial oxygen consumption and increase oxygen supply.

More

It relaxes vascular smooth muscle, releases nitric oxide (NO) through metabolism to produce isosorbide mononitrate (ISMN), activates guanylate cyclase, and increases cyclic guanosine monophosphate (cGMP) in smooth muscle cells, thereby relaxing vascular smooth muscle, dilating peripheral arteries and veins, reducing the amount of blood returning to the heart, lowering left ventricular end-diastolic pressure and pulmonary capillary wedge pressure (preload), reducing peripheral vascular resistance, systolic arterial pressure and mean arterial pressure (afterload), dilating coronary arteries and increasing coronary perfusion. The overall effect is to reduce myocardial oxygen consumption and increase oxygen supply.

87-33-2 23
Isosorbide dinitrate for injection
Isosorbide dinitrate
Name Description Content CAS NO. Registered Holders
Isosorbide dinitrate

It relaxes vascular smooth muscle, releases nitric oxide through metabolism to produce isosorbide mononitrate, activates guanylate cyclase, and increases cyclic guanosine monophosphate in smooth muscle cells, thereby relaxing vascular smooth muscle, dilating peripheral arteries and veins, reducing the amount of blood returning to the heart, lowering left ventricular end-diastolic pressure and pulmonary capillary wedge pressure, reducing peripheral vascular resistance, systolic arterial pressure and mean arterial pressure, dilating coronary arteries to increase coronary perfusion, and ultimately reducing myocardial oxygen consumption and increasing oxygen supply.

More

It relaxes vascular smooth muscle, releases nitric oxide through metabolism to produce isosorbide mononitrate, activates guanylate cyclase, and increases cyclic guanosine monophosphate in smooth muscle cells, thereby relaxing vascular smooth muscle, dilating peripheral arteries and veins, reducing the amount of blood returning to the heart, lowering left ventricular end-diastolic pressure and pulmonary capillary wedge pressure, reducing peripheral vascular resistance, systolic arterial pressure and mean arterial pressure, dilating coronary arteries to increase coronary perfusion, and ultimately reducing myocardial oxygen consumption and increasing oxygen supply.

87-33-2 23
Isosorbide dinitrate for injection
Isosorbide dinitrate
Name Description Content CAS NO. Registered Holders
Isosorbide dinitrate

It relaxes vascular smooth muscle, releases nitric oxide through metabolism to produce isosorbide mononitrate, activates guanylate cyclase, and increases cyclic guanosine monophosphate in smooth muscle cells, thereby relaxing vascular smooth muscle, dilating peripheral arteries and veins, reducing the amount of blood returning to the heart, lowering left ventricular end-diastolic pressure and pulmonary capillary wedge pressure, reducing peripheral vascular resistance, systolic arterial pressure and mean arterial pressure, increasing coronary perfusion, reducing myocardial oxygen consumption and increasing oxygen supply.

More

It relaxes vascular smooth muscle, releases nitric oxide through metabolism to produce isosorbide mononitrate, activates guanylate cyclase, and increases cyclic guanosine monophosphate in smooth muscle cells, thereby relaxing vascular smooth muscle, dilating peripheral arteries and veins, reducing the amount of blood returning to the heart, lowering left ventricular end-diastolic pressure and pulmonary capillary wedge pressure, reducing peripheral vascular resistance, systolic arterial pressure and mean arterial pressure, increasing coronary perfusion, reducing myocardial oxygen consumption and increasing oxygen supply.

87-33-2 23
Feedback & Suggestions
Send Message

Thank you for your feedback. If you require further assistance, please contact us by email at info@echemi.com or call us at +86-532-55729510.