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Founded in:
1989-11-08 -
Country:
China -
Address:
No. 1288, Shitai Road, Baoshan District, Shanghai -
Tax NO.:
913101131322006102 -
Registered Funds:
105.05 million yuan -
Website:
-
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Sodium bicarbonate |
This product is an antacid. After oral administration, it can quickly neutralize stomach acid and relieve symptoms of excessive stomach acid or heartburn, but its effect is weak and lasts for a short time.
More
This product is an antacid. After oral administration, it can quickly neutralize stomach acid and relieve symptoms of excessive stomach acid or heartburn, but its effect is weak and lasts for a short time. |
144-55-8 | 45 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Salvia Root P.E Tanshinone IIA 20% |
|
0 | ||
| Fermented Cordyceps powder |
|
0 | ||
| PERSICAE SEMEN |
|
0 | ||
| Pine Pollen Disruption Powder |
|
0 | ||
| GYNOSTEMMA PENTAPHYLLUM P.E. |
|
0 | ||
| Schisandra chinensis (processed) |
|
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Salvia Root P.E Tanshinone IIA 20% |
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine
More
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine |
0 | ||
| Fermented Cordyceps powder |
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine
More
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine |
0 | ||
| PERSICAE SEMEN |
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine
More
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine |
0 | ||
| Pine Pollen Disruption Powder |
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine
More
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine |
0 | ||
| GYNOSTEMMA PENTAPHYLLUM P.E. |
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine
More
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine |
0 | ||
| Schisandra chinensis (processed) |
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine
More
Inhibits the degree of liver fibrosis in rats induced by carbon tetrachloride plus high-fat diet; inhibits the increase of serum alanine aminotransferase in rats induced by carbon tetrachloride and D-galactosamine |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Thiamine chloride |
It is an important component of coenzymes required for sugar metabolism
More
It is an important component of coenzymes required for sugar metabolism |
0.2mg | 59-43-8 | 9 |
| Riboflavin |
An important coenzyme component required for tissue respiration
More
An important coenzyme component required for tissue respiration |
0.1mg | 83-88-5 | 19 |
| Ascorbic Acid |
Participates in the formation of antibodies, collagen and tissue repair in the body, as well as the metabolism of sugar, fat and protein
More
Participates in the formation of antibodies, collagen and tissue repair in the body, as well as the metabolism of sugar, fat and protein |
2.5mg | 50-81-7 | 28 |
| Vitamin D3 |
Participate in the metabolism of calcium and phosphorus and promote new bone formation
More
Participate in the metabolism of calcium and phosphorus and promote new bone formation |
70unit | 67-97-0 | 23 |
| CALCIUM GLUCONATE MONOHYDRATE |
Calcium ions are necessary for maintaining normal function of nerves, muscles and bones, and also play an important role in the body's coagulation mechanism and reducing capillary permeability.
More
Calcium ions are necessary for maintaining normal function of nerves, muscles and bones, and also play an important role in the body's coagulation mechanism and reducing capillary permeability. |
150mg | 66905-23-5 | 35 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Carbamazepine |
Anticonvulsant, anti-epileptic, anti-neuropathic pain, anti-manic-depressive, improve the symptoms of certain mental illnesses, anti-central diabetes insipidus. Possible mechanisms include use-dependent blocking of various excitable cell membrane Na channels, inhibition of T-type calcium channels, enhancement of central noradrenergic nerve activity, promotion of antidiuretic hormone (ADH) secretion or increase in effector sensitivity to ADH.
More
Anticonvulsant, anti-epileptic, anti-neuropathic pain, anti-manic-depressive, improve the symptoms of certain mental illnesses, anti-central diabetes insipidus. Possible mechanisms include use-dependent blocking of various excitable cell membrane Na channels, inhibition of T-type calcium channels, enhancement of central noradrenergic nerve activity, promotion of antidiuretic hormone (ADH) secretion or increase in effector sensitivity to ADH. |
298-46-4 | 42 |