Centrally acting muscle relaxants that act on multiple synaptic pathways in the central nervous system to produce muscle relaxants

Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism and reduces capillary permeability, etc.

This product is a bone metabolism regulator. It has a strong affinity for calcium phosphate in the body, can inhibit abnormal calcification and excessive bone absorption in the human body, relieve bone pain, reduce the concentration of serum alkaline phosphatase and urinary hydroxyproline; at low doses, it can directly inhibit osteoclast formation and prevent bone absorption, reduce bone turnover rate, increase bone density, etc. to achieve bone calcium regulation.

1. Stimulate the pancreatic islet beta cells to secrete insulin, which is a prerequisite for the islet beta cells to also have a certain ability to synthesize and secrete insulin; 2. Inhibit liver glycogenolysis and gluconeogenesis by increasing portal vein insulin levels or directly acting on the liver, and reduce liver glucose production and output; 3. It may also increase the sensitivity of extra-pancreatic tissues to insulin and the utilization of sugar (probably mainly through post-receptor effects). Therefore, the overall effect is to lower fasting blood sugar and postprandial blood sugar.

Regulate autonomic dysfunction and endocrine imbalance