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Founded in:
1993-07-05 -
Country:
China -
Address:
The First Pharmaceutical Park, Economic and Technological Development Zone, Datong City, Shanxi Province -
Tax NO.:
91140200602165291J -
Registered Funds:
395 million yuan -
Website:
-
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| ATP disodium salt |
It can penetrate the cell membranes of various important organs, increase the ATP level in tissues and cells, and directly supply energy to cells under ischemic and hypoxic conditions, thereby improving cell energy, metabolism and cell membrane sodium pump mechanism, reducing cell swelling, improving microcirculatory disorders, and restoring organ function; it can enter the mitochondria of ischemic and hypoxic myocardial cells, support cell function, improve the imbalance of sodium, potassium, and magnesium inside and outside the cells, inhibit the aggregation of intracellular Ca2+, have a certain effect of scavenging oxygen free radicals, protect cells, and promote functional recovery.
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It can penetrate the cell membranes of various important organs, increase the ATP level in tissues and cells, and directly supply energy to cells under ischemic and hypoxic conditions, thereby improving cell energy, metabolism and cell membrane sodium pump mechanism, reducing cell swelling, improving microcirculatory disorders, and restoring organ function; it can enter the mitochondria of ischemic and hypoxic myocardial cells, support cell function, improve the imbalance of sodium, potassium, and magnesium inside and outside the cells, inhibit the aggregation of intracellular Ca2+, have a certain effect of scavenging oxygen free radicals, protect cells, and promote functional recovery. |
0.1g | 987-65-5 | 8 |
| Magnesium chloride |
Improve cellular energy metabolism, improve microcirculation disorders, improve macrophage function; improve rat liver cell membrane permeability caused by shock, effectively restore ischemic liver cell function, improve the imbalance of sodium, potassium, and magnesium inside and outside the cells, inhibit intracellular Ca2+ aggregation, protect cells, and promote functional recovery.
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Improve cellular energy metabolism, improve microcirculation disorders, improve macrophage function; improve rat liver cell membrane permeability caused by shock, effectively restore ischemic liver cell function, improve the imbalance of sodium, potassium, and magnesium inside and outside the cells, inhibit intracellular Ca2+ aggregation, protect cells, and promote functional recovery. |
32mg | 7786-30-3 | 25 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Ginkgo biloba |
Regulate vascular tension, antagonize PAF, inhibit platelet aggregation, reduce blood viscosity, increase blood flow to ischemic organs, improve microcirculation and blood rheology, remove free radicals, reduce damage from harmful substances, and protect nerve cells.
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Regulate vascular tension, antagonize PAF, inhibit platelet aggregation, reduce blood viscosity, increase blood flow to ischemic organs, improve microcirculation and blood rheology, remove free radicals, reduce damage from harmful substances, and protect nerve cells. |
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| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Levothyroxine sodium |
1. Increase the effect of anticoagulants; 2. Increase the level of phenytoin sodium in the blood; 3. Anticonvulsants such as carbamazepine and phenytoin sodium accelerate its metabolism and can displace thyroxine from plasma proteins; 4. The dosage of cardiac glycosides needs to be adjusted when used with cardiac glycosides; 5. Increase the effect of sympathomimetic drugs; 6. Increase the sensitivity of catecholamine receptors, thereby increasing the response of tricyclic antidepressants; 7. Cholestyramine reduces its absorption, and its dosage needs to be increased when taking oral contraceptives at the same time.
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1. Increase the effect of anticoagulants; 2. Increase the level of phenytoin sodium in the blood; 3. Anticonvulsants such as carbamazepine and phenytoin sodium accelerate its metabolism and can displace thyroxine from plasma proteins; 4. The dosage of cardiac glycosides needs to be adjusted when used with cardiac glycosides; 5. Increase the effect of sympathomimetic drugs; 6. Increase the sensitivity of catecholamine receptors, thereby increasing the response of tricyclic antidepressants; 7. Cholestyramine reduces its absorption, and its dosage needs to be increased when taking oral contraceptives at the same time. |
55-03-8 | 15 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Nimodipine |
Nimodipine is a Ca2 channel blocker that inhibits smooth muscle contraction and relieves vascular spasm by preventing Ca2 from entering cells. It has a strong effect on cerebral arteries, can pass through the blood-brain barrier, increase cerebral blood flow, reduce ischemic brain damage, and also has the effect of protecting and promoting memory and promoting intellectual recovery.
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Nimodipine is a Ca2 channel blocker that inhibits smooth muscle contraction and relieves vascular spasm by preventing Ca2 from entering cells. It has a strong effect on cerebral arteries, can pass through the blood-brain barrier, increase cerebral blood flow, reduce ischemic brain damage, and also has the effect of protecting and promoting memory and promoting intellectual recovery. |
66085-59-4 | 17 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Nimodipine |
Extract from the above information
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Extract from the above information |
66085-59-4 | 17 |