The antipyretic and analgesic effects are faster and stronger than those of aminopyrine.

Inhibits T lymphocyte activation, combines with intracellular protein FKBP-12 to form a complex, inhibits the phosphatase activity of calcineurin, prevents the dephosphorylation and translocation of NF-AT, inhibits the transcription of multiple genes and the release of mediators, and downregulates the expression of FCRI on the surface of Langerhans cells.

Inhibits T lymphocyte activation, combines with FKBP-12 to form a complex to inhibit calcineurin activity, prevents dephosphorylation and translocation of NF-AT, inhibits the transcription of multiple lymphokine genes (such as IL-2, IL-3, IL-4, IL-5, GM-CSF, TNF-?), inhibits the release of mediators in skin mast cells and basophils, and downregulates FCRI expression on the surface of Langerhans cells.

This product is a macrolide antibiotic, which has inhibitory effects on Gram-positive bacteria such as Staphylococcus aureus, Streptococcus, and Pneumococcus, as well as some Gram-negative bacteria such as Haemophilus influenzae, Bordetella pertussis, Neisseria gonorrhoeae, Legionella pneumophila, and some anaerobic bacteria such as Bacteroides fragilis, Streptococcus peptostreptococcus, and Propionibacterium acnes. In addition, it also has inhibitory effects on mycoplasma. The characteristics of this product are that its antibacterial activity in vitro is similar to that of erythromycin, but its antibacterial activity in vivo against some bacteria such as Staphylococcus aureus, Streptococcus, and Haemophilus influenzae is stronger than that of erythromycin. There is cross-resistance between this product and erythromycin. The mechanism of action of this product is to inhibit the association of the 50S subunit of the nuclear protein and inhibit protein synthesis to produce an antibacterial effect.

It enhances the body's disease resistance by regulating the body's cellular immunity, humoral immunity, stimulating the reticuloendothelial system, activating the function of monocytes and macrophages, and enhancing the function of natural killer cells; it achieves anti-allergic and anti-asthmatic effects by stabilizing mast cells, blocking IgE function, reducing the release of active substances from degranulated cells, and resisting bronchospasm caused by acetylcholine.

It enhances the body's disease resistance by regulating the body's cellular immunity, humoral immunity, stimulating the reticuloendothelial system, activating the function of monocytes and macrophages, and enhancing the function of natural killer cells; it achieves anti-allergic and anti-asthmatic effects by stabilizing mast cells, blocking IgE function, reducing the release of active substances from degranulated cells, and resisting bronchospasm caused by acetylcholine.