It is an antiemetic drug of the 5-HT3 receptor antagonist class. It has a strong and selective antagonistic effect on 5-HT3 receptors. It has a high affinity for 5-HT3 receptors. Its action strength is about 410 times stronger than metoclopramide, 2 times stronger than ondansetron, and almost equal to gressilon. It can highly antagonize bradycardia induced by 5-HT. Its antagonistic effect is about 900 times stronger than metoclopramide, about 4 times stronger than ondansetron, and equal to gressilon. In in vitro tests on sensory nerve cells, isolated hearts or isolated ileum, it shows an antagonistic effect on 5-HT3 receptors. This effect is 20 to 720 times stronger than metoclopramide and 1/8 to 3 times stronger than gressilon.

When used topically and long-term, it can stimulate hair growth in people with male pattern baldness and alopecia areata. The exact mechanism by which it works to treat hair loss is unknown.

Minoxidil is a peripheral vasodilator that can stimulate hair growth in patients with male pattern baldness and alopecia areata when used topically for a long time. The exact mechanism by which this product treats hair loss is unknown. Clinical studies have shown that when used topically in normotensive and untreated hypertensive patients, no systemic effects due to absorption of minoxidil were observed.

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This product is a potent, selective non-steroidal aromatase inhibitor. It can inhibit the conversion of androstenedione produced in the adrenal glands of postmenopausal patients into estrone, thereby significantly reducing plasma estrogen levels and inhibiting breast tumor growth. In addition, this product has no significant effect on the production of adrenal cortical steroids or aldosterone.