It binds to dopamine receptors in the hypothalamus, transmits electrical impulses from the hypothalamus to the spinal cord through the paraventricular nucleus of the hypothalamus, which controls sexual desire, and promotes blood flow to the penis. At the same time, it affects the activation of nitric oxide, causing it to be absorbed and then transported to the brain, causing a series of chain reactions in the human body, and finally increasing blood flow and causing penile erection.

It binds to dopamine receptors in the hypothalamus, transmits electrical impulses from the hypothalamus to the spinal cord through the paraventricular nucleus of the hypothalamus, which controls sexual desire, and promotes blood flow to the penis. At the same time, it affects the activation of nitric oxide, causing it to be absorbed and then transported to the brain, causing a series of chain reactions in the human body, and finally increasing blood flow and causing penile erection.

Nucleoside antiviral drugs. After entering the cell, this product is rapidly phosphorylated to a monophosphate compound, and then becomes a triphosphate compound through the action of cellular kinase. Its phosphorylation in cells infected with cytomegalovirus is faster than that in normal cells. Ganciclovir can competitively inhibit DNA polymerase and incorporate into the DNA of viruses and host cells, thereby inhibiting DNA synthesis. This product has a stronger inhibitory effect on viral DNA polymerase than host cell polymerase. In animal experiments, this product has teratogenic, carcinogenic, immunosuppressive effects and reproductive system toxicity.

Nucleoside antiviral drugs. After entering the cell, this product is rapidly phosphorylated to a monophosphate compound, and then becomes a triphosphate compound through the action of cellular kinase. Its phosphorylation in cells infected with cytomegalovirus is faster than that in normal cells. Ganciclovir can competitively inhibit DNA polymerase and incorporate into the DNA of viruses and host cells, thereby inhibiting DNA synthesis. This product has a stronger inhibitory effect on viral DNA polymerase than host cell polymerase. In animal experiments, this product has teratogenic, carcinogenic, immunosuppressive effects and reproductive system toxicity.

Antiviral drug. It has an inhibitory effect on herpes simplex virus, varicella zoster virus, cytomegalovirus, etc. in vitro. After entering the cells infected by herpes virus, it competes with deoxynucleoside for viral thymidine kinase or cell kinase, and the drug is phosphorylated into activated acyclovir triphosphate, which inhibits viral replication by interfering with viral DNA polymerase and binding to the growing DNA chain to cause the extension of the DNA chain to be interrupted. It has a special affinity for viruses, but has low toxicity to mammalian host cells.