Antiviral drug. It has an inhibitory effect on herpes simplex virus, varicella zoster virus, cytomegalovirus, etc. in vitro. After entering the cells infected by herpes virus, this product competes with deoxynucleoside for viral thymidine kinase or cell kinase, and the drug is phosphorylated into activated acyclovir triphosphate, and then inhibits viral replication in two ways: ① Interfering with viral DNA polymerase and inhibiting viral replication; ② Under the action of DNA polymerase, it binds to the growing DNA chain, causing the extension of the DNA chain to be interrupted. This product has a special affinity for viruses, but has low toxicity to mammalian host cells.

Ciclopirox is a broad-spectrum antifungal drug that has antifungal effects on pathogenic fungi that can cause nail fungus infections.

It has high activity against protozoa and anaerobic bacteria, and can inhibit most anaerobic bacteria; microaerophilic bacteria and Helicobacter pylori are sensitive to it; the MIC against Trichomonas vaginalis is similar to that of metronidazole. Its metabolites are more active against Gardnerella than metronidazole. The mechanism of action of this product is to reduce it into a cytotoxin, thereby acting on the DNA metabolism process of bacteria and promoting bacterial death. Drug-resistant bacteria often lack nitroreductase and are resistant to this product. The mechanism of anti-amoeba is to inhibit its redox reaction.

This product is a medium-acting non-depolarizing muscle relaxant of the monoquaternary ammonium steroid class. It blocks the conduction between nerve endings and striated muscles by competing with acetylcholine for nicotinic receptors located at the motor end plate of striated muscles. Intravenous injection of 0.08-0.1mg/kg produces significant effects within 1 minute, reaches a peak in 3-5 minutes, and lasts for 30-90 minutes. The muscle relaxant effect is 3 times stronger than that of tubocurarine chloride; it has no vagal nerve blocking effect and is suitable for patients with myocardial ischemia and heart disease, but the use of vagal excitatory drugs and β-receptor blockers is prone to bradycardia. This product has a weak histamine release effect, and can also cause bronchospasm and allergic reactions, but they are rare.

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