This product is an immunomodulatory drug. It has the function of regulating and enhancing the cellular immune function of the human body. It can promote the maturation of T lymphocytes in peripheral blood after mitogen activation, increase the secretion of various lymphokines (such as α, γ interferon, interleukin 2 and interleukin 3) after T cells are activated by various antigens or mitogens, and increase the level of lymphokine receptors on T cells. It also enhances lymphocyte response by activating T4 helper cells. In addition, this product may affect the chemotaxis of NK precursor cells, which become more cytotoxic after exposure to interferon. Therefore, this product has the function of regulating and enhancing the cellular immune function of the human body. This product is an immune bidirectional modulator. It has the function of inducing and promoting the differentiation, maturation and activation of T lymphocytes and their subsets, and regulating T lymphocytes.

This drug is an immune enhancement drug. It is a polynucleotide peptide (molecular weight less than 5000) extracted from healthy human blood or animal spleen. It is not easily destroyed by RNAse, DNAse and trypsin. This drug is an important factor in cellular immune response. It has no antigenicity and is not an antibody fragment, but it can transmit the cellular immune reactivity of multiple antigens, including bacteria, fungi, viruses, tissue compatibility antigens and tumor-specific antigens, etc. It can enhance the immune reactivity of receptor cells, promote the chemotaxis of macrophages, increase delayed cell response, and improve the body's ability to kill tumors.

It binds to the viral DNA polymerase, reducing its activity and inhibiting DNA synthesis. After phosphorylation, it generates adenosine arabinoside diphosphate (Ara-ADP) and adenosine arabinoside triphosphate (Ara-ATP). Ara-ATP and deoxyadenosine triphosphate (dATP) compete with each other to bind to viral DNAP, thereby inhibiting the activity of the enzyme and the synthesis of viral DNA. It also inhibits the activity of viral nucleotide reductase and inhibits the synthesis of viral DNA. It can also inhibit the activity of viral DNA terminal deoxynucleotidyl transferase, allowing Ara-A to penetrate into the viral DNA and connect to the end of the DNA chain at the 3'-OH position, inhibiting the continued synthesis of viral DNA.

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