YiChang SanXia Pharmaceutical Co., Ltd.

Prevent nitrogen loss, correct negative nitrogen balance and reduce protein consumption

Prevent nitrogen loss, correct negative nitrogen balance and reduce protein consumption

Prevent nitrogen loss, correct negative nitrogen balance and reduce protein consumption

Prevent nitrogen loss, correct negative nitrogen balance and reduce protein consumption

Aminoglycoside antibiotics have antibacterial effects on most Enterobacteriaceae, Pseudomonas aeruginosa, some other Pseudomonas, Acinetobacter, Alcaligenes, meningococci, gonococci, influenza bacilli, Yersinia, Campylobacter fetus, Mycobacterium tuberculosis and some Mycobacterium. The mechanism of action is to act on the 30S subunit of bacterial ribosomes and inhibit bacterial protein synthesis. Combination with semi-synthetic penicillins or cephalosporins can achieve synergistic antibacterial effects.

Vitamin B6 is converted into pyridoxal phosphate in red blood cells and acts as a coenzyme in various metabolic functions of proteins, carbohydrates, and lipids. It is also involved in the conversion of tryptophan into niacin or 5-hydroxytryptamine.

This product has a broad-spectrum antimicrobial effect in vitro, including aerobic Gram-negative and Gram-positive bacteria, anaerobes, Rickettsia, spirochetes and Chlamydia. It has bactericidal effects on the following bacteria: influenza bacillus, Streptococcus pneumoniae and Neisseria meningitidis. It only has antibacterial effects on the following bacteria: Staphylococcus aureus, Streptococcus pyogenes, Streptococcus viridans, group B hemolytic Streptococcus, Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Salmonella typhi, Salmonella paratyphi, Shigella, Bacteroides fragilis and other anaerobic bacteria. The following bacteria are usually resistant to chloramphenicol: Pseudomonas aeruginosa, Acinetobacter, Enterobacter, Serratia marcescens, indole-positive Proteus, methicillin-resistant Staphylococcus and Enterococcus. This product is an antibacterial agent. Chloramphenicol is fat-soluble and enters bacterial cells by diffusion, and reversibly binds to the 50S subunit of the bacterial ribosome, which blocks the growth of the peptide chain (possibly due to the inhibition of the action of transpeptidase), thereby inhibiting the formation of peptide chains and preventing protein synthesis.

Sorbitol monosaccharide is an isomer of mannitol, and its effect is similar to that of mannitol but weaker. After intravenous injection of concentrated solution of this product, except for a small part converted into sugar, most of it is excreted through the kidney in its original form. Due to the formation of blood hypertonicity, it can dehydrate the surrounding tissues and brain parenchyma and be excreted from the urine with the drug, thereby reducing intracranial pressure and eliminating edema. High efficiency appears 2 hours after injection, and the brain edema is gradually calmed down, the tension disappears, the cerebrospinal fluid pressure decreases, it is not metabolized in the body, and is rarely reabsorbed in the renal tubules after glomerular filtration, playing an osmotic diuretic role. 1 Tissue dehydration effect. Increase plasma colloidal osmotic pressure, causing water in tissues (including eyes, brain, cerebrospinal fluid, etc.) to enter the blood vessels, thereby reducing tissue edema, reducing intraocular pressure, intracranial pressure and cerebrospinal fluid capacity.