It has anti-myocardial ischemia and anti-hypoxia effects and enhances immune function

It has anti-myocardial ischemia and anti-hypoxia effects and enhances immune function

It has anti-myocardial ischemia and anti-hypoxia effects and enhances immune function

It is a peripheral analgesic. Its mechanism of action is to inhibit the synthesis and release of prostaglandins (PGE1) in the hypothalamic temperature regulation center, thereby causing peripheral vasodilation and sweating to achieve an antipyretic effect. At the same time, it can inhibit the effects of PGE1, bradykinin and histamine, increase the pain threshold and produce analgesic effect.

It can inhibit the synthesis and release of prostaglandins in the hypothalamus, restore the normal responsiveness of the receptor neurons in the body temperature regulation center and have an antipyretic effect; at the same time, it also has an analgesic effect by inhibiting the synthesis of prostaglandins, etc., and can inhibit the synthesis and release of prostaglandins in local inflammatory tissues, stabilize lysosomal enzymes, and affect the phagocytic function of macrophages to have an anti-inflammatory effect.

It is a central nervous system stimulant that can excite the cerebral cortex, increase sensitivity to the outside world, constrict cerebral blood vessels, and enhance the effect of the first two drugs in relieving headaches.

It has a strong histamine H1 receptor blocking effect, can alleviate other respiratory symptoms caused by allergies, and also has a mild inhibitory effect on the central nervous system.

This product is a nitrofuran antibacterial drug. It has certain antibacterial effects on both Gram-positive and Gram-negative bacteria, including Salmonella, Shigella, Escherichia coli, Klebsiella pneumoniae, Enterobacter, Staphylococcus aureus, Enterococcus faecalis, Streptococcus pyogenes, Vibrio cholerae, Campylobacter, Bacteroides, etc. It is also active against Trichomonas and Giardia at a certain concentration. Its mechanism of action is to interfere with bacterial oxidoreductase and thus block the normal metabolism of bacteria.

Trimethoprim (TMP) is an antibacterial agent, a lipophilic weak base, and its chemical structure belongs to the pyrimethamine class. It has antibacterial activity against Escherichia coli, Klebsiella, Proteus mirabilis, Salmonella, and Shigella, but has no obvious antibacterial effect on Streptococcus pneumoniae, Neisseria gonorrhoeae, and Neisseria meningitidis, and has no effect on Pseudomonas aeruginosa. The mechanism of action of this product is to interfere with bacterial folic acid metabolism. It mainly selectively inhibits the activity of bacterial dihydrofolate reductase, so that dihydrofolate cannot be reduced to tetrahydrofolate, and synthetic folic acid is the main component of nucleic acid biosynthesis. Therefore, this product prevents the synthesis of bacterial nucleic acids and proteins, and the binding of this product to bacterial dihydrofolate reductase is 50,000 to 60,000 times tighter than that to mammalian enzymes. The combination of this product and sulfonamides can double block the bacterial folic acid synthesis metabolism, have a synergistic effect, enhance the antibacterial activity of sulfonamides, and convert the antibacterial effect into a bactericidal effect, reducing the production of drug-resistant strains.