This product is a derivative of benzimidazole. It has a strong affinity for the beta-tubulin of the insect body and can bind to it at very low concentrations, thereby inhibiting the polymerization of microtubules, causing the disappearance of the epidermis or intestinal cells of the insect body, reducing digestion and reducing the absorption of nutrients such as glucose, leading to the death of the insect body. This drug can also inhibit fumarate reductase in mitochondria, reduce glucose transport, and uncouple oxidative phosphorylation, thereby affecting the production of ATP. This drug has effects on both adults and eggs.

Itraconazole is a triazole derivative with broad-spectrum antifungal activity. In vitro tests have shown that it can inhibit the growth of a variety of human pathogenic fungi within a conventional concentration range, including dermatophytes, yeasts, Aspergillus, etc. It exerts its antifungal effect by destroying the synthesis of ergosterol in the fungal cell membrane.

A selective monoamine antagonist with high affinity for 5HT2 receptors, D2 receptors, alpha 1 and alpha 2 receptors, and H1 receptors. It also has antagonistic effects on other receptors, but they are weaker. It has low to moderate affinity for 5HT1C, 5HT1D, and 5HT1A, weak affinity for D1 and haloperidol-sensitive sigma receptors, and no affinity for M receptors or beta 1 and beta 2 receptors. It is believed that its therapeutic effect is the result of the combined effect of antagonism on D2 receptors and 5HT2 receptors. The antagonism of receptors other than D2 and 5HT2 may be related to other effects of risperidone.

A selective monoamine antagonist with high affinity for 5HT2 receptors, D2 receptors, α1 and α2 receptors, and H1 receptors. It also has antagonistic effects on other receptors, but they are weaker. It has low to moderate affinity for 5HT1C, 5HT1D, and 5HT1A, weak affinity for D1 and haloperidol-sensitive σ receptors, and no affinity for M receptors or β1 and β2 receptors. It is believed that its therapeutic effect is the result of the combined effect of antagonism on D2 receptors and 5HT2 receptors. The antagonism of receptors other than D2 and 5HT2 may be related to other effects of risperidone.

Antifungal drugs, which have antibacterial activity against dermatophytes, molds and yeasts (such as Candida), and are also effective against some Gram-positive bacteria

Glucocorticoids, which have anti-inflammatory, antipruritic and anti-allergic effects