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Founded in:
1997-12-30 -
Country:
China -
Address:
Wangtai, Jincheng City, Shanxi Province -
Tax NO.:
91140500111208832X -
Registered Funds:
148.512131 yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Bifidobacterium longum culture medium (B) |
It forms a joint flora with Lactobacillus acidophilus and Enterococcus faecalis, forms a biological barrier on the surface of the intestinal mucosa, prevents the invasion of pathogenic bacteria and inhibits the production of endotoxins, thereby maintaining the normal physiological function of the intestine.
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It forms a joint flora with Lactobacillus acidophilus and Enterococcus faecalis, forms a biological barrier on the surface of the intestinal mucosa, prevents the invasion of pathogenic bacteria and inhibits the production of endotoxins, thereby maintaining the normal physiological function of the intestine. |
0 | ||
| lactobacillus acidophilus |
It forms a joint flora with Bifidobacterium longum and Enterococcus faecalis, forming a biological barrier on the surface of the intestinal mucosa, preventing the invasion of pathogenic bacteria and inhibiting the production of endotoxins, thereby maintaining the normal physiological function of the intestine.
More
It forms a joint flora with Bifidobacterium longum and Enterococcus faecalis, forming a biological barrier on the surface of the intestinal mucosa, preventing the invasion of pathogenic bacteria and inhibiting the production of endotoxins, thereby maintaining the normal physiological function of the intestine. |
308084-36-8 | 1 | |
| Enterococcus faecalis |
It forms a combined flora with Bifidobacterium longum and Lactobacillus acidophilus, forming a biological barrier on the surface of the intestinal mucosa, preventing the invasion of pathogenic bacteria and inhibiting the production of endotoxins, thereby maintaining the normal physiological function of the intestine.
More
It forms a combined flora with Bifidobacterium longum and Lactobacillus acidophilus, forming a biological barrier on the surface of the intestinal mucosa, preventing the invasion of pathogenic bacteria and inhibiting the production of endotoxins, thereby maintaining the normal physiological function of the intestine. |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Otilonium bromide |
Extract from the above information
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Extract from the above information |
26095-59-0 | 4 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Otilonium bromide |
Spamamine belongs to a class of antispasmodic and anticholinergic drugs. It has a selective and strong antispasmodic effect on the smooth muscle of the digestive tract. It is suitable for all hypermotility, spasmodic reactions caused by different causes and different parts, and pathological atrophy of smooth muscle fibers.
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Spamamine belongs to a class of antispasmodic and anticholinergic drugs. It has a selective and strong antispasmodic effect on the smooth muscle of the digestive tract. It is suitable for all hypermotility, spasmodic reactions caused by different causes and different parts, and pathological atrophy of smooth muscle fibers. |
26095-59-0 | 4 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Sodium ferulic |
The results of animal studies show that sodium ferulate can inhibit the production of malondialdehyde and thromboxane B2, reduce myocardial edema and the release of lactate dehydrogenase, and promote the production of 6-keto-prostaglandin F1α, and has anti-platelet aggregation, vasodilation and myocardial protection effects.
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The results of animal studies show that sodium ferulate can inhibit the production of malondialdehyde and thromboxane B2, reduce myocardial edema and the release of lactate dehydrogenase, and promote the production of 6-keto-prostaglandin F1α, and has anti-platelet aggregation, vasodilation and myocardial protection effects. |
24276-84-4 | 8 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Propyl gallate |
1. It can obviously inhibit the synthesis of thromboxane A2 (TXA2), counteract the platelet aggregation caused by arachidonic acid (AA), and has a stronger and faster antiplatelet aggregation effect than aspirin (ASP). 2. It can enhance the fibrinolytic activity and promote the dissolution of thrombus. 3. It can reduce the specific viscosity of whole blood and plasma, and accelerate the electrophoresis of red blood cells. 4. It can relax the vascular smooth muscle, dilate the artery, and increase the blood flow of the coronary artery and cerebral artery. 5. It can improve the ability to resist hypoxia. 6. It can strongly scavenge free radicals. 7. It can inhibit the inflammatory exudation of capillaries.
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1. It can obviously inhibit the synthesis of thromboxane A2 (TXA2), counteract the platelet aggregation caused by arachidonic acid (AA), and has a stronger and faster antiplatelet aggregation effect than aspirin (ASP). 2. It can enhance the fibrinolytic activity and promote the dissolution of thrombus. 3. It can reduce the specific viscosity of whole blood and plasma, and accelerate the electrophoresis of red blood cells. 4. It can relax the vascular smooth muscle, dilate the artery, and increase the blood flow of the coronary artery and cerebral artery. 5. It can improve the ability to resist hypoxia. 6. It can strongly scavenge free radicals. 7. It can inhibit the inflammatory exudation of capillaries. |
121-79-9 | 10 |