It can directly stimulate adrenaline receptors, and can also indirectly stimulate adrenaline receptors by prompting adrenaline nerve endings to release norepinephrine. It has a stimulating effect on both alpha and beta receptors. It can dilate bronchi and constrict local blood vessels, and its action time is longer; it strengthens myocardial contractility, increases cardiac output, and makes venous return to the heart sufficient; it has a stronger central nervous system stimulating effect than adrenaline.

It has ① antihistamine effect, which can compete with histamine released from tissues for H1 receptors on effector cells, thereby stopping allergic reactions; ② at the same time, it inhibits central nervous system activity and causes sedative and hypnotic effects; ③ it enhances the effect of antitussive drugs.

This product is a nucleoside antiviral drug that has a selective inhibitory effect on a variety of herpes viruses (HSV). After acyclovir enters HSV-infected cells, it binds to the specific thymidylate kinase encoded by the virus and is rapidly converted into acyclosine monophosphate, which is then converted into acyclosine diphosphate by the cell guanylate kinase, and then converted into acyclosine triphosphate by other cell enzymes, and competes with guanylate triphosphate to infect herpes simplex virus DNA polymerase, thereby inhibiting viral DNA synthesis. Acyclovir triphosphate is a strong viral DNA polymerase inhibitor, which has an inhibitory effect on the DNA polymerase of five human herpes viruses, namely HSV-1, HSV-2, VZV, CMZ and EBV, and has no effect on normal cells. Its antiviral efficacy is 160 times stronger than that of adenosine.

Reserpine is an adrenergic neuron blocking antihypertensive drug. It achieves antihypertensive, heart rate slowing and central nervous system inhibition effects by depleting adrenaline in peripheral sympathetic nerve endings, catecholamines and 5-hydroxytryptamine in the heart, brain and other tissues. The antihypertensive effect is mainly achieved by reducing cardiac output and peripheral resistance, and partially inhibiting cardiovascular reflexes. The effect of slowing the heart rate is not obvious in people with normal heart rate, but it is obvious in people with sinus tachycardia. Reserpine acts on the hypothalamus to produce a sedative effect, but does not cause drowsiness or anesthetic effects, does not change the electroencephalogram during sleep, and can relieve anxiety, tension and headaches in hypertensive patients.

This product is an adrenal cortex hormone drug. It has multiple pharmacological effects such as anti-inflammatory, anti-allergic and immunosuppressive. Its main mechanisms are: 1. Anti-inflammatory effect: Glucocorticoids reduce and prevent tissue response to inflammation, thereby reducing the manifestation of inflammation. 2. Anti-allergic and immunosuppressive effects: prevent or inhibit intermediate immune responses, delayed allergic reactions, and reduce the expansion of primary immune responses.