Creatine phosphate plays an important role in the energy metabolism of muscle contraction. It is the chemical energy reserve of myocardial and skeletal muscles and is used for the resynthesis of ATP. The hydrolysis of ATP provides energy for the contraction process of actomyosin. Insufficient energy supply caused by slowed oxidative metabolism is an important factor in the formation and development of myocardial cell damage. Insufficient creatine phosphate levels have important clinical significance in the damage of myocardial contractility and functional recovery ability. In fact, in myocardial injury, there is a close relationship between the amount of high-energy phosphate compounds in cells and the survival and contractile function recovery ability of cells. Therefore, maintaining the level of high-energy phosphate compounds has become the basic principle of various methods to limit myocardial damage, and it is also the basis for cardiac metabolic protection. Animal experiments and human cardiac arrest experiments have shown the role of sodium creatine phosphate and its possibility of protecting the myocardium.

Creatine phosphate plays an important role in the energy metabolism of muscle contraction. It is the chemical energy reserve of myocardial and skeletal muscles and is used for the resynthesis of ATP. The hydrolysis of ATP provides energy for the contraction process of actomyosin. Insufficient energy supply caused by slowed oxidative metabolism is an important factor in the formation and development of myocardial cell damage. Insufficient creatine phosphate levels have important clinical significance in the damage of myocardial contractility and functional recovery ability. Maintaining the level of high-energy phosphate compounds has become the basic principle of various methods to limit myocardial damage, and it is also the basis for cardiac metabolic protection. Animal experiments and human cardiac arrest experiments have shown the role of sodium creatine phosphate and its possibility of protecting the myocardium.

Extract from the above information

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After being absorbed by the digestive tract, it is converted into active metabolites in the body. Its active metabolites exert analgesic, anti-inflammatory and antipyretic effects by inhibiting the synthesis of prostaglandins.