By inhibiting cyclooxygenase, it selectively inhibits the synthesis of prostaglandins in the hypothalamic temperature regulation center, leading to peripheral vasodilation and sweating to achieve an antipyretic effect; by inhibiting the synthesis and release of prostaglandins, it increases the pain threshold and has an analgesic effect. It is a peripheral analgesic, weaker than aspirin, and is only effective for mild and moderate pain. This product has no obvious anti-inflammatory effect.

By inhibiting cyclooxygenase, it selectively inhibits the synthesis of prostaglandins in the hypothalamic temperature regulation center, leading to peripheral vasodilation and sweating to achieve the effect of antipyretic; by inhibiting the synthesis and release of prostaglandins, it increases the pain threshold and plays an analgesic role. It is a peripheral analgesic, with a stronger effect than acetaminophen, and is only effective for mild and moderate pain. This product has no obvious anti-inflammatory effect.

No specific pharmacological action is directly mentioned.

Not yet clear

It can directly stimulate adrenaline receptors, and can also indirectly stimulate adrenaline receptors by prompting adrenaline nerve endings to release norepinephrine. It has a stimulating effect on both alpha and beta receptors. It can dilate bronchi and constrict local blood vessels, and its action time is longer; it strengthens myocardial contractility, increases cardiac output, and makes venous return to the heart sufficient; it has a stronger central nervous system stimulating effect than adrenaline.

It has ① antihistamine effect, which can compete with histamine released from tissues for H1 receptors on effector cells, thereby stopping allergic reactions; ② at the same time, it inhibits central nervous system activity and causes sedative and hypnotic effects; ③ it enhances the effect of antitussive drugs.

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

Azorrole antifungal drugs have antibacterial and bactericidal effects on dermatophytes, yeasts, dimorphic fungi and fungi. The mechanism of action is to highly selectively interfere with the activity of fungal cytochrome P-450, thereby inhibiting the biosynthesis of ergosterol on the fungal cell membrane.

A powerful topical corticosteroid that acts rapidly and has strong capillary constriction and anti-inflammatory effects.

Ketoconazole is an azole antifungal drug. It has antibacterial and bactericidal effects on dermatophytes, yeasts (Candida, Pityrosporum, Torulopsis, Cryptococcus), dimorphic fungi and Fungi; except for Entomophthora, it has weaker effects on Aspergillus, Schenckia, some Phaeochromocytaceae and Mucor. The mechanism of action of ketoconazole is mainly to highly selectively interfere with the activity of fungal cytochrome P-450, thereby inhibiting the biosynthesis of ergosterol on the fungal cell membrane. Clobetasol propionate is a potent topical corticosteroid. It acts quickly and has strong capillary constriction and anti-inflammatory effects.