Because this product has the physical and chemical characteristics of strong negative charge, it can interfere with many links of the blood coagulation process and has anticoagulant effect in vivo and in vitro. Its mechanism of action is relatively complex, mainly through binding with antithrombin III (AT-III), thereby enhancing the latter's inhibitory effect on activated coagulation factors II, IX, X, XI and XII. Its consequences involve preventing platelet aggregation and destruction, hindering the formation of coagulation activating enzymes; preventing prothrombin from turning into thrombin; inhibiting thrombin, thereby preventing fibrinogen from turning into fibrin.

This product is a vitamin drug. Vitamin B6 is converted into pyridoxal phosphate in red blood cells and acts as a coenzyme in various metabolic functions of proteins, carbohydrates, and lipids. It also participates in the conversion of tryptophan into niacin or 5-hydroxytryptamine.

This product is a vitamin drug. Vitamin B6 is converted into pyridoxal phosphate in red blood cells and acts as a coenzyme in various metabolic functions of proteins, carbohydrates, and lipids. It also participates in the conversion of tryptophan into niacin or 5-hydroxytryptamine.

It can bind to the DNA of malarial parasites to form a complex that inhibits DNA replication and RNA transcription, thereby inhibiting the protein synthesis of the parasites; it reduces the oxygen consumption of malarial parasites, inhibits the phosphorylase in malarial parasites and interferes with their sugar metabolism; it causes hemochromatin aggregation and is accompanied by cell death; a certain concentration can cause premature rupture of parasitized red blood cells and prevent the maturation of schizonts. It is ineffective in the erythrocyte stage, and a long course of treatment can cure malignant malaria, but it has no direct effect on the gametocytes of malignant malaria and cannot interrupt transmission.

Small doses act on the higher centers of the cerebral cortex, promoting mental excitement; larger doses have the effect of stimulating the respiratory center and vasomotor center of the medulla oblongata.

It has a strong effect on most Gram-positive and Gram-negative bacteria. It has a good antibacterial effect on Escherichia coli, Klebsiella pneumoniae, Proteus vulgaris, Shigella, and Salmonella, and its activity is slightly worse than kanamycin. It also has a good effect on some Staphylococci, gonococci, and meningococci. It has a weak effect on Streptococcus and Mycobacterium tuberculosis, and is ineffective against Pseudomonas aeruginosa and anaerobic bacteria. This product is cross-resistant with kanamycin. The results of the ototoxicity test on guinea pigs in toxicological experiments showed that this product caused significantly less hearing and cochlear pathological damage to guinea pigs than kanamycin and gentamicin.