This product is a semi-synthetic ergot alkaloid derivative. It has α receptor blocking and vasodilation effects. It can enhance the metabolism of brain cell energy and increase the utilization of oxygen and glucose. It can promote the conversion of neurotransmitter dopamine to enhance nerve conduction, strengthen brain protein biosynthesis, and improve brain function. Toxicity tests failed to show that nicergoline has teratogenic effects.

It has α receptor blocking and vasodilation effects. It can enhance brain cell energy metabolism and increase the utilization of oxygen and glucose. It can promote the conversion of the neurotransmitter dopamine to enhance nerve conduction, strengthen brain protein biosynthesis, and improve brain function. Toxicity tests have failed to show that nicergoline has teratogenic effects.

It can reduce the increase of serum AST and ALT in the animal acute liver injury model caused by TAA (thioacetamide) and CC14 (carbon tetrachloride), and inhibit the accumulation of triglycerides caused by chronic liver injury model; it can promote liver glycogen synthesis, inhibit the increase of cholesterol, and help to restore the serum albumin/globulin ratio.

The semi-synthetic product obtained from vascopine has the effect of reducing and breaking mucopolysaccharide fibers in sputum, reducing sputum viscosity, making sputum thinner and easier to cough up. Secondly, it can inhibit the synthesis of acidic glycoproteins in mucous glands and goblet cells, reducing the content of sialic acid (one of the components of acidic mucopolysaccharides) in sputum, reducing sputum viscosity, and facilitating sputum coughing up. The expectorant effect of this drug is also related to its promotion of ciliary movement of the respiratory mucosa and its malignant expectorant effect. As sputum is coughed up, the patient's ventilation can be improved.

It has a strong affinity for the liver's steroid metabolic enzymes, thereby hindering the inactivation of cortisol and aldosterone, showing obvious corticosteroid-like effects (such as anti-inflammatory, anti-allergic and protective membrane structure), without obvious corticosteroid-like adverse reactions; promoting monochromatic metabolism, reducing the release of ALT and AST; inducing γ-IFN and interleukin II, increasing NK cell activity and the OKT4/OKT8 ratio and activating the reticuloendothelial system; inhibiting the release of histamine from mast cells; inhibiting the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reactions; inhibiting the production and formation of free radicals and lipid peroxides, reducing the activity of proline hydroxylase; regulating calcium ion channels, protecting lysosomal membranes and mitochondria

Glycyrrhizic acid monoamine has a strong affinity for steroid metabolic enzymes in the liver, thereby hindering the inactivation of cortisol and aldosterone. After use, it shows obvious corticosteroid-like effects, such as anti-inflammatory effects, anti-allergic effects, and protective membrane structures; there are no obvious corticosteroid-like adverse reactions. This product can promote monochrome metabolism, reduce the release of ALT and AST; induce γ-IFN and interleukin II, increase NK cell activity and OKT4/OKT8 ratio and activate the reticuloendothelial system; inhibit mast cell release of histamine; inhibit the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reactions; inhibit the production and formation of free radicals and lipid peroxides, and reduce the activity of proline hydroxylase; regulate calcium ion channels, protect lysosomal membranes and mitochondria

Glycyrrhizic acid monoamine has a strong affinity for steroid metabolic enzymes in the liver, thereby hindering the inactivation of cortisol and aldosterone. After use, it shows obvious corticosteroid-like effects, such as anti-inflammatory effects, anti-allergic effects, and protective membrane structures; there are no obvious corticosteroid-like adverse reactions. This product can promote monochrome metabolism, reduce the release of ALT and AST; induce γ-IFN and interleukin II, increase NK cell activity and OKT4/OKT8 ratio and activate the reticuloendothelial system; inhibit mast cell release of histamine; inhibit the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reactions; inhibit the production and formation of free radicals and lipid peroxides, and reduce the activity of proline hydroxylase; regulate calcium ion channels, protect lysosomal membranes and mitochondria