Citalopram is an antidepressant and a dicyclic hydrogenated phthalide derivative. Its antidepressant mechanism may be related to inhibiting the reuptake of 5-HT by neurons in the central nervous system, thereby enhancing the function of central serotonergic nerves. In vitro and animal experiments indicate that citalopram is a highly selective 5-HT reuptake inhibitor with little effect on the reuptake of norepinephrine and dopamine. Rats were given citalopram for 14 days, and the effect of inhibiting 5-HT uptake was not tolerated. Citalopram is a racemic body, and its effect of inhibiting 5-HT reuptake is mainly exerted by its (S)-enantiomer. Citalopram has no obvious affinity for 5-HT1A, 5-HT2A, D1 receptor, D2 receptor, а1 receptor, а2 receptor, β receptor, H1 receptor, and GABA.

It has antibacterial effects on Gram-positive and -negative aerobic and anaerobic bacteria, and is very sensitive to Streptococcus pneumoniae, Streptococcus pyogenes, Staphylococcus aureus (including enzyme-producing strains), Escherichia coli, Klebsiella, some strains of Acinetobacter, Bacteroides fragilis and other Bacteroides, Peptococcus and some strains of Peptostreptococcus. It is also quite sensitive to Streptococcus faecalis, Streptococcus epidermidis, Haemophilus influenzae, Proteus mirabilis, Serratia marcescens, Enterobacter aerogenes, Enterobacter cloacae, Pseudomonas aeruginosa, Clostridium difficile, Clostridium difficile, etc. It has good enzyme resistance and rarely has cross-resistance with other beta-lactam drugs.

Imipenem has antibacterial effects on Gram-positive and Gram-negative aerobic and anaerobic bacteria. This product is very sensitive to Streptococcus pneumoniae, Streptococcus pyogenes, Staphylococcus aureus (including enzyme-producing strains), Escherichia coli, Klebsiella, some strains of Acinetobacter, Bacteroides fragilis and other Bacteroides, Peptococcus and some strains of Peptostreptococcus. This product is also quite sensitive to Streptococcus faecalis, Streptococcus epidermidis, Haemophilus influenzae, Proteus mirabilis, Serratia marcescens, Enterobacter aerogenes, Enterobacter cloacae, Pseudomonas aeruginosa, Clostridium difficile, Clostridium difficile, etc. This product has good enzyme resistance and rarely has cross-resistance with other beta-lactam drugs.

Competitive M-cholinergic receptor blocker, used to relieve symptoms of frequent urination, urgency and urgency incontinence caused by overactive bladder. It has high selectivity for M-cholinergic receptors, strong anticholinergic activity, and weak effects or affinity on receptors and potential cellular targets of other neurotransmitters.

This product is a benzimidazole compound. After oral absorption, it is transferred to the gastric mucosa and converted into an active metabolite under acidic conditions. The activated body specifically inhibits the H/K-ATPase system of gastric parietal cells and blocks the final step of gastric acid secretion. This product inhibits basal gastric acid secretion and gastric acid secretion under stimulation in a dose-dependent manner. This product has no antagonistic effect on choline and amine H2 receptors.