Not clearly described

Non-nucleoside reverse transcriptase inhibitors (NNRTIs) of human immunodeficiency virus (HIV-1) bind directly to the reverse transcriptase (RT) of HIV-1 and block both RNA-dependent and DNA-dependent DNA polymerase activity by cleaving the catalytic end of the enzyme without competing with substrates or nucleoside triphosphates.

This product is a drug that inhibits the synthesis of uric acid. Allopurinol and its metabolite oxypurinol can inhibit xanthine oxidase, preventing hypoxanthine and xanthine from being metabolized into uric acid, thereby reducing the production of uric acid. It reduces the uric acid content in the blood and urine to a level below the solubility, prevents uric acid from forming crystals and depositing in joints and other tissues, and also helps to redissolve uric acid crystals in the tissues of gout patients. Allopurinol also inhibits the synthesis of new purines in the body by acting on hypoxanthine-guanine phosphate nucleic acid convertase.

This product is a sulfonamide antibiotic. When used in combination with trimethoprim, it can double-block the bacterial folic acid metabolism and has a good antibacterial effect on a variety of bacteria.

It works by inhibiting dihydrofolate reductase and can be used in combination with sulfamethoxazole to enhance the synergistic antibacterial effect and reduce drug-resistant strains.

It has astringent, gastric mucosal protective and antibacterial effects. Because this product is insoluble in water, it forms a protective film on the gastric mucosal wound surface after oral administration, reducing the irritation of food to the gastric mucosa; in the intestine, bismuth combines with hydrogen sulfide to form insoluble bismuth sulfide (Bi2S3) on the intestinal mucosa, slowing down intestinal peristalsis; at the same time, bismuth salts also have antibacterial effects.