The drug can cause necrosis of the intrauterine decidual tissue and produce endogenous prostaglandins, which can cause uterine contractions. Ethacridine also has a direct excitatory effect on uterine muscles.

This product is a total alkaloid preparation of Rauwolfia ovata, and its main component is reserpine. Its antihypertensive effect is similar to that of reserpine, but its effect is milder than that of reserpine. Reserpine depletes the storage of norepinephrine in the peripheral sympathetic nerve terminals, and also depletes the storage of catecholamines and 5-hydroxytryptamine in the brain, heart and other organs. The antihypertensive effect is mainly peripheral, and the peripheral vascular resistance is reduced, the heart rate is slowed down, and the cardiac output is reduced, thereby lowering blood pressure.

It has a bidirectional regulatory effect on the intestine, and can antagonize the tonic contraction of the intestine caused by acetylcholine and barium chloride, relax the intestine and relieve spasm; it can also antagonize the relaxation of the intestine caused by adrenaline, so that the intestine can resume spontaneous activity. It has no obvious effect on the spontaneous activity of the isolated small intestine of mice, but can enhance the spontaneous activity of the isolated small intestine of rabbits and dogs. It has no effect on the gastric bioelectricity of normal rabbits, that is, it does not affect the normal gastrointestinal motility function.

Monk fruit mainly has a bidirectional regulatory effect on the intestine. It antagonizes the tonic contraction of the intestine caused by acetylcholine and barium chloride, relaxes the intestine and relieves spasms. It also antagonizes the relaxation of the intestine caused by adrenaline, restores spontaneous activity of the intestine, indicating that it has a bidirectional regulatory effect on the motor function of the intestine. Monk fruit has no obvious effect on the spontaneous activity of the isolated small intestine of mice, but can enhance the spontaneous activity of the isolated small intestine of rabbits and dogs. Monk fruit has no effect on the gastric bioelectricity of normal rabbits, that is, it does not affect the normal motor function of the digestive tract.

Monk fruit mainly has a bidirectional regulatory effect on the intestine. It antagonizes the tonic contraction of the intestine caused by acetylcholine and barium chloride, relaxes the intestine and relieves spasms. It also antagonizes the relaxation of the intestine caused by adrenaline, restores spontaneous activity of the intestine, indicating that it has a bidirectional regulatory effect on the motor function of the intestine. Monk fruit has no obvious effect on the spontaneous activity of the isolated small intestine of mice, but can enhance the spontaneous activity of the isolated small intestine of rabbits and dogs. Monk fruit has no effect on the gastric bioelectricity of normal rabbits, that is, it does not affect the normal motor function of the digestive tract.

Monk fruit mainly has a bidirectional regulatory effect on the intestine. It antagonizes the tonic contraction of the intestine caused by acetylcholine and barium chloride, relaxes the intestine and relieves spasms. It also antagonizes the relaxation of the intestine caused by adrenaline, restores spontaneous activity of the intestine, indicating that it has a bidirectional regulatory effect on the motor function of the intestine. Monk fruit has no obvious effect on the spontaneous activity of the isolated small intestine of mice, but can enhance the spontaneous activity of the isolated small intestine of rabbits and dogs. Monk fruit has no effect on the gastric bioelectricity of normal rabbits, that is, it does not affect the normal motor function of the digestive tract.

Monk fruit mainly has a bidirectional regulatory effect on the intestine. It antagonizes the tonic contraction of the intestine caused by acetylcholine and barium chloride, relaxes the intestine and relieves spasms. It also antagonizes the relaxation of the intestine caused by adrenaline, restores spontaneous activity of the intestine, indicating that it has a bidirectional regulatory effect on the motor function of the intestine. Monk fruit has no obvious effect on the spontaneous activity of the isolated small intestine of mice, but can enhance the spontaneous activity of the isolated small intestine of rabbits and dogs. Monk fruit has no effect on the gastric bioelectricity of normal rabbits, that is, it does not affect the normal motor function of the digestive tract.

It has analgesic, sedative, hypnotic and tranquilizing effects. Its analgesic effect is weaker than pethidine, but stronger than general antipyretic analgesics. It has no respiratory depression effect at therapeutic doses and does not cause gastrointestinal smooth muscle spasm. It has a better effect on chronic persistent pain and dull visceral pain, but has a poor effect on acute sharp pain (such as postoperative pain, traumatic pain, etc.) and advanced cancer pain. While producing analgesic effects, it can also cause sedation and hypnosis.