It has an inhibitory effect on Japanese encephalitis virus, mumps virus, influenza virus, etc.; it has a significant antipyretic effect on fever in experimental animals caused by typhoid and cholera mixed vaccines; it can enhance the phagocytic effect of white blood cells on bacteria and increase the phagocytic index.

It has an inhibitory effect on many kinds of bacteria and can also delay the pathogenic effects of respiratory viruses on cells.

Experimental studies have shown that this prescription has a wide range of pharmacological effects, good antibacterial and antiviral effects, and no obvious toxic and adverse reactions. Pharmacological experiments have shown that Daqingye, honeysuckle, ginseng, and rhubarb have significant inhibitory effects on Staphylococcus aureus, hemolytic streptococci, Pseudomonas aeruginosa, and Escherichia coli. Daqingye has an inhibitory effect on Japanese encephalitis virus, mumps virus, influenza virus, etc. Enema with Daqingye decoction has a significant antipyretic effect on experimental animals with fever caused by typhoid and cholera mixed vaccines, and can enhance the phagocytic effect of leukocytes on bacteria and improve the phagocytic index. Honeysuckle has an inhibitory effect on a variety of bacteria and can also delay the pathological effects of respiratory viruses on cells. Rhubarb has an antibacterial effect on most Gram-positive bacteria and some Gram-negative bacteria. It can enhance the phagocytic activity of leukocytes on Staphylococcus aureus and significantly accelerate the phagocytic process of leukocytes.

It has significant inhibitory effects on Staphylococcus aureus, hemolytic Streptococcus, Pseudomonas aeruginosa, Escherichia coli, etc.

It has antibacterial effect on most Gram-positive bacteria and some Gram-negative bacteria; it can enhance the phagocytic activity of leukocytes against Staphylococcus aureus and can significantly accelerate the phagocytic process of leukocytes.

Broad-spectrum antiviral drug. It has the effect of inhibiting the growth of multiple viruses such as respiratory syncytial virus, influenza virus, hepatitis A virus, adenovirus, etc. in vitro, and its mechanism is not fully understood. After entering the virus-infected cells, the drug is rapidly phosphorylated, and its product acts as a competitive inhibitor of viral synthase, inhibiting inosine monophosphate dehydrogenase, influenza virus RNA polymerase and mRNA guanosine transferase, thereby causing a decrease in intracellular guanosine triphosphate, impairing viral RNA and protein synthesis, and inhibiting viral replication and transmission. It may also have an immune effect and neutralizing antibody effect on respiratory syncytial virus.