Wanbangde Pharmaceutical Group Co., Ltd.

This product is a fluoroquinolone antibacterial drug. It has strong antibacterial effects on Gram-negative bacteria, including Escherichia coli, Klebsiella pneumoniae, Proteus, Salmonella typhi, Salmonella paratyphi, Shigella, Enterobacter cloacae, Enterobacter aerogenes, Citrobacter, Serratia marcescens, Pseudomonas aeruginosa, Neisseria meningitidis, Haemophilus influenzae, Moracata, Legionella pneumophila, Neisseria gonorrhoeae, etc. It also has moderate antibacterial effects on Gram-positive cocci such as Staphylococcus and hemolytic Streptococcus. In recent years, bacterial resistance to fluoroquinolones has increased significantly, especially in Escherichia coli, with a resistance rate of more than 50%. The mechanism of action of this product is to inhibit the bacterial DNA gyrase and thus kill bacteria.

This product is a fluoroquinolone antibiotic that has antibacterial effects on Gram-negative bacteria (such as Escherichia coli, Klebsiella pneumoniae, etc.) and some Gram-positive cocci (such as Staphylococcus aureus and hemolytic Streptococcus). Its mechanism is to inhibit the bacterial DNA gyrase and achieve bactericidal effect.

1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc. It mainly inhibits the active reabsorption of NaCl by the thick wall segment of the renal tubular loop, reduces the osmotic pressure gradient difference of the medullary interstitial fluid, and leads to increased excretion of water, Na, and Cl-; it promotes the secretion of K by the distal tubules. It inhibits the reabsorption of Ca2+ and Mg2+ by the loop of Henle and increases their excretion. Short-term medication can increase uric acid excretion, while long-term medication can cause hyperuricemia. 2. Effect on hemodynamics: Furosemide can inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, dilate renal blood vessels, reduce renal vascular resistance, and increase renal blood flow; it is of great significance in diuresis. In addition, it can dilate pulmonary capacitance veins, reduce pulmonary capillary permeability, reduce ventricle return blood volume, and reduce left ventricular end-diastolic pressure, which is helpful for the treatment of acute left heart failure and provides a theoretical basis for the treatment of adult respiratory distress syndrome.

Promote uterine contraction, hematopoiesis and anti-anemia effects, improve microcirculation, anti-inflammatory

Contraction of the uterus, analgesia, anti-platelet aggregation and anti-thrombotic effects, improvement of microcirculation, anti-inflammatory

Promote uterine contraction, anti-platelet aggregation and anti-thrombotic effects, improve microcirculation

Anti-platelet aggregation and anti-thrombotic effects, promote uterine contraction, improve microcirculation

A new generation of macrolide antibiotics, it mainly acts on Gram-positive bacteria, anaerobic bacteria, Chlamydia and Mycoplasma, etc. Its in vitro antibacterial effect is similar to that of erythromycin, and its in vivo antibacterial effect is 1 to 4 times stronger than that of erythromycin.