Kaifeng Pharmaceutical (GROUP) Co., Ltd.

1. Treat metabolic acidosis. This product can increase the bicarbonate concentration in plasma, neutralize hydrogen ions, and thus correct acidosis. 2. Alkalize urine. When using this product, the bicarbonate concentration in urine increases, and the pH of urine increases, making it difficult for uric acid, sulfonamides, and hemoglobin to form crystals or aggregate in urine. 3. Antacid. Oral administration can quickly neutralize or buffer gastric acid without directly affecting gastric acid secretion. Therefore, the pH in the stomach increases rapidly, relieving symptoms caused by high gastric acid. Pharmacokinetics: This product can be excreted by the kidneys in the form of bicarbonate, and can also be excreted from the body by the lungs in the form of carbon dioxide.

1. Treat metabolic acidosis. This product can increase the bicarbonate concentration in plasma, neutralize hydrogen ions, and thus correct acidosis. 2. Alkalize urine. When this product is used, the bicarbonate concentration in urine increases, and the pH of urine increases, making it difficult for uric acid, sulfonamides, and hemoglobin to form crystals or aggregate in urine. 3. Antacid. Oral administration can quickly neutralize or buffer gastric acid without directly affecting gastric acid secretion. As a result, the pH in the stomach increases rapidly, relieving symptoms caused by high gastric acid. Pharmacokinetics: This product can be excreted by the kidneys in the form of bicarbonate, and can also be excreted from the body by the lungs in the form of carbon dioxide.

1. Treat metabolic acidosis. This product can increase the bicarbonate concentration in plasma, neutralize hydrogen ions, and thus correct acidosis. 2. Alkalize urine. As the bicarbonate concentration in urine increases, the pH of urine increases, making it difficult for uric acid, sulfonamides, and hemoglobin to form crystals or aggregate in urine. 3. Antacid. Oral administration can quickly neutralize or buffer gastric acid without directly affecting gastric acid secretion. The pH in the stomach increases rapidly, relieving symptoms caused by high gastric acid. In terms of pharmacokinetics, this product can be excreted by the kidneys in the form of bicarbonate, or it can be excreted from the body from the lungs in the form of carbon dioxide.

1. Treat metabolic acidosis. This product can increase the bicarbonate concentration in plasma, neutralize hydrogen ions, and thus correct acidosis. 2. Alkalize urine. When this product is used, the bicarbonate concentration in urine increases, and the pH of urine increases, making it difficult for uric acid, sulfonamides, and hemoglobin to form crystals or aggregate in urine. 3. Antacid. Oral administration can quickly neutralize or buffer gastric acid without directly affecting gastric acid secretion. As a result, the pH in the stomach increases rapidly, relieving symptoms caused by high gastric acid. Pharmacokinetics: This product can be excreted by the kidneys in the form of bicarbonate, and can also be excreted from the body by the lungs in the form of carbon dioxide.

Extract from the above information

It has anti-epithelial cell proliferation, epithelial cell differentiation induction and anti-inflammatory effects; it blocks the pathological signal substances expressed by abnormal immune system in psoriasis by inhibiting the activity of protein kinase C in keratinocytes and polymorphonuclear leukocytes, inhibiting the secretion of interleukin-1, 6, 8 and tumor necrosis factor-α by human peripheral blood monocytes; it reduces the amount of transforming growth factor-α in keratinocytes and its affinity with epidermal growth factor receptor, thereby producing anti-keratinocyte proliferation and anti-inflammatory effects; it causes proliferation of mouse tail granular layer cells and down-regulates the expression of transglutaminase (TGase) mRNA, indicating that it has the effect of inducing epithelial cell differentiation; it produces anti-inflammatory effects by inhibiting the production of reactive oxygen species by polymorphonuclear leukocytes induced by phorbol esters.

It has anti-epithelial cell proliferation, epithelial cell differentiation induction and anti-inflammatory effects. It blocks the pathological signal substances expressed by abnormal immune system in psoriasis by inhibiting the activity of protein kinase C in keratinocytes and polymorphonuclear leukocytes, inhibiting the secretion of interleukin-1, 6, 8 and tumor necrosis factor α by human peripheral blood monocytes; and reduces the amount of transforming growth factor-α in keratinocytes and its affinity with epidermal growth factor receptor, thereby producing anti-keratinocyte proliferation and anti-inflammatory effects. It can cause proliferation of mouse tail granular cells and down-regulate the expression of transglutaminase (TGase) mRNA, indicating that it has the effect of inducing epithelial cell differentiation. It produces anti-inflammatory effects by inhibiting the production of reactive oxygen species by polymorphonuclear leukocytes induced by phorbol esters.