This product is a coenzyme that improves the body's metabolism and participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotides in the body. It is also the main source of energy in the body. When energy is needed for absorption, secretion, muscle contraction and biochemical synthesis reactions in the body, adenosine triphosphate is decomposed into adenosine diphosphate and phosphate groups, and energy is released at the same time. Animal experiments have shown that this product can inhibit the slow calcium ion influx of slow-reacting fibers, block or delay the forward conduction in the atrioventricular node reentry pathway, and a large dose may also block or delay the forward and reverse conduction of the bypass; in addition, it also has a short-term and strong effect of enhancing the vagus nerve, thereby terminating arrhythmias caused by atrioventricular node reentry and bypass reentry mechanisms.

Netilmicin is a semi-synthetic aminoglycoside broad-spectrum antibiotic. Its mechanism of action is to inhibit the normal protein synthesis of sensitive microorganisms. This product has a wide range of antimicrobial effects, mainly targeting Gram-negative rods and a few Gram-positive bacteria, including Citrobacter, Enterobacter, Escherichia coli, Klebsiella, Morgan Proteus, Pseudomonas aeruginosa, Salmonella, Shigella and Staphylococcus (penicillin- and methicillin-resistant). This product is also active in vitro against some isolated Acinetobacter, Neisseria, indole-positive Proteus, Pseudomonas, and Serratia. Most streptococci and anaerobic microorganisms such as Clostridium are resistant to aminoglycosides.

Non-digitalis cardiotonic agent with positive inotropic effect, can enhance myocardial contractility, improve cardiac pumping function, dilate blood vessels, reduce myocardial oxygen consumption; improve myocardial cell metabolism, protect ischemic and hypoxic myocardium; can improve sinoatrial node P cell function.

Non-digitalis cardiotonic agent with positive inotropic effect, can enhance myocardial contractility, improve cardiac pumping function, dilate blood vessels, reduce myocardial oxygen consumption; improve myocardial cell metabolism, protect ischemic and hypoxic myocardium; can improve sinoatrial node P cell function.

Non-digitalis cardiotonic agents have positive inotropic effects, can enhance myocardial contractility, improve myocardial pump function, have vasodilatory effects, can reduce myocardial oxygen consumption; improve myocardial cell metabolism, protect ischemic and hypoxic myocardium; can improve sinoatrial node P cell function.

Cyclic adenosine meglumine is a non-digitalis cardiotonic agent with positive inotropic effect. It can enhance myocardial contractility, improve myocardial pump function, dilate blood vessels, and reduce myocardial oxygen consumption. It can improve myocardial cell metabolism and protect ischemic and hypoxic myocardium. It can improve the function of sinoatrial node P cells.