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I. Skeletal Muscle Regeneration and Repair (Core Function, Superior to Ordinary MGF)
Long-lasting activation of muscle satellite stem cells
In vitro muscle cell models continuously awaken dormant satellite cells, promoting the division of stem cells and their integration into damaged muscle fibers; PEG modification extends the duration of action, increasing the cell repair cycle, and providing more stable repair effects for minor muscle tears caused by high-intensity exercise.
Significantly enhance myoprotein synthesis efficiency
Upregulate the mTOR synthesis pathway, accelerate amino acid absorption and muscle fiber protein deposition; simultaneously inhibit the ubiquitination protein degradation pathway, reducing muscle loss, and better maintaining muscle reserves in models of calorie deficit and aging sarcopenia cells.
Mechanically directed local muscle fiber hypertrophy
Only effective on muscle tissues subjected to tension and mechanical load, precisely promoting the thickening of muscle fibers in the stressed area; will not indiscriminately stimulate the uncontrolled proliferation of soft tissues throughout the body, used for local muscle shaping and research on the mechanism of exercise-induced muscle growth.
Shorten the fatigue recovery period of muscle cells
Inhibit the release of TNF-α and IL-6 inflammatory damage factors after exercise, reduce oxidative stress and lactic acid accumulation in muscle cells, alleviate muscle pain-related cellular inflammatory damage.
II. Mitochondrial Energy Metabolism Enhancement
Promote the birth of skeletal muscle mitochondria
Upregulate the PGC-1α mitochondrial synthesis gene, increase the number of mitochondria in muscle cells, improve the efficiency of aerobic oxidation for energy supply, and enhance the endurance reserve of muscle cells.
Stabilize the anti-apoptosis of mitochondrial membranes
Remove reactive oxygen species produced by muscle metabolism, maintain the balance of mitochondrial membrane potential, block the programmed cell death of muscle cells induced by oxidation and hypoxia, and reduce muscle cell loss caused by long-term training.
Optimize the conversion of sugar and lipid energy supply
Increase the expression of glucose transporter proteins on muscle cell membranes, accelerate sugar intake; simultaneously promote the oxidation and decomposition of fat in the muscle, balance energy supply, and delay the occurrence of cellular fatigue.
III. Tendon, Cartilage, and Connective Tissue Repair (In vitro Joint Tissue)
Enhance cartilage matrix synthesis
Stimulate type II collagen and glycosaminoglycan secretion in vitro by chondrocytes, reduce cartilage cell apoptosis caused by mechanical wear and inflammation, used for basic research on joint degeneration in exercise.
Strengthen tendon and ligament fiber toughness
Activate tendon fibroblast proliferation, increase collagen fiber secretion, improve the elasticity of connective tissue caused by strain, and reduce the risk of fibrosis and sclerosis.
Basic maintenance of bone cells
Slightly promote osteoblast activity, assist in bone matrix mineralization, inhibit excessive breakdown of bone by osteoclasts, used for in vitro screening of bone repair in exercise-induced injuries.
IV. Broad-spectrum Cellular Antioxidant Protection of Multiple Organs
Cardiovascular endothelium and myocardium protection
Stabilize mitochondrial of vascular endothelial cells, reduce lipid oxidation deposition signals; in ischemic myocardial models, reduce the proportion of myocardial cell apoptosis, alleviate hypoxic injury.
Neuron repair
Act on isolated neurons, remove oxidative free radicals in the brain, stabilize mitochondrial of neurons, reduce neuronal aging and apoptosis, used for research on the mechanism of exercise-induced brain fatigue and nerve injury.
Skin fibroblast in vitro repair
Promote collagen secretion of dermal cells, resist collagen degradation caused by ultraviolet rays and oxidation; accelerate the migration of epidermal cells, only for in vitro observation of cell experiments on wound surfaces, no transdermal external use effective data.
Hazard Identification
Classification of the substance or mixture
GHS label elements, including precautionary statements
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Other hazards which do not result in classification
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Handling and Storage
Precautions for safe handling
Handling in a well ventilated place. Wear suitable protective clothing. Avoid contact with skin and eyes. Avoid formation of dust and aerosols. Use non-sparking tools. Prevent fire caused by electrostatic discharge steam.
Conditions for safe storage, including any incompatibilities
Store the container tightly closed in a dry, cool and well-ventilated place. Store apart from foodstuff containers or incompatible materials.
United States
