Edifenphos

310.37

310.37

241-178-1

770M9U0F8Q

DTXSID7041910

Yellow to light brown liquid... .|Clear yellow to light-brown liquid

2930909058

76.9

3.48

Yellow to brown liquid with a characteristic odor.

1.251 g/cm³ @ Temp: 20 °C

-25 °C

154 °C @ Press: 8 x 10^-3 Torr

214ºC

nD22 1.61

In water, 0.056 mg/mL at 20 °C

0-6°C

1.3 x 10 -2 Pa (20 °C)

Oral-rat LD50: 100 mg/kg; Oral-Mouse LD50: 143 mg/kg

Open flame is flammable; heated to decompose toxic phosphorus oxide, sulfur oxide, nitrogen oxide gas

Characteristic odor

III

6.1(b)

UN 2810

3

21-23/25-43-50/53-24/25-23

36/37-45-60-61

TE3850000

T,N

The warehouse is ventilated, low temperature and dry; stored and transported separately from food materials

Stable in neutral media. Hydrolysed by strong acids and alkalis; at 25 deg C, DT50 19 days (pH 7), 2 days (pH 9). Degradable in the presence of light.

P261-P273-P280-P301 + P310-P311-P501

H301 + H311 + H331-H317-H410

SRP: The most favorable course of action is to use an alternative chemical product with less inherent propensity for occupational exposure or environmental contamination. Recycle any unused portion of the material for its approved use or return it to the manufacturer or supplier. Ultimate disposal of the chemical must consider: the material's impact on air quality; potential migration in soil or water; effects on animal, aquatic, and plant life; and conformance with environmental and public health regulations.

Incompatible with alkaline materials.

WHO (2001) The Who Recommended Classification of Pesticides by Hazard and Guidelines to Classification 2000-2002 (WHO/PC/01.5), International Programme on Chemical Safety

|Danger|H301: Toxic if swallowed [Danger Acute toxicity, oral]|P261, P264, P270, P271, P272, P273, P280, P301+P310, P302+P352, P304+P340, P311, P312, P321, P322, P330, P333+P313, P363, P391, P403+P233, P405, and P501|H301 (100%): Toxic if swallowed [Danger Acute toxicity, oral]|Aggregated GHS information provided by 130 companies from 2 notifications to the ECHA C&L Inventory. Each notification may be associated with multiple companies.|P260, P261, P264, P270, P271, P280, P301+P310, P302+P352, P304+P340, P305+P351+P338, P309+P311, P311, P312, P314, P321, P322, P330, P332+P313, P337+P313, P361, P363, P403+P233, P405, and P501

If material on fire or involved in fire: Do not extinguish fire unless flow can be stopped. Use water in flooding quantities as fog. Solid streams of water may be ineffective. Cool all affected containers with flooding quantities of water. Apply water from as far a distance as possible. Use "alcohol" foam, dry chemical or carbon dioxide. /Organophosphorus pesticides, liquid, flammable, toxic; Organophosphorus pesticides, liquid, toxic/|If material on fire or involved in fire: Use water in flooding quantities as fog. Extinguish fire using agent suitable for type of surrounding fire. (Material itself does not burn or burns with difficulty.) /Organophosphorus pesticides, solid, toxic/

Environmental considerations: Air spill: Apply water spray or mist to knock down vapors. /Organophosphorus pesticides, liquid, flammable, toxic; Organophosphorus pesticides, liquid, toxic; Organophosphorus pesticides, solid, toxic/|Environmental considerations: Water spill: Use natural barriers or oil spill control booms to limit spill travel. Remove trapped material with suction hoses. /Organophosphorus pesticides, liquid, flammable, toxic; Organophosphorus pesticides, liquid, toxic; Organophosphorus pesticides, solid, toxic/|Environmental considerations: Land spill: Dig a pit, pond, lagoon, holding area to contain liquid or solid material. /SRP: If time permits, pits, ponds, lagoons, soak holes, or holding areas should be sealed with an impermeable flexible membrane liner./ Dike surface flow using soil, sand bags, foamed polyurethane, or foamed concrete. Absorb bulk liquid with fly ash, cement powder, or commercial sorbents. /Organophosphorus pesticides, liquid, flammable, toxic; Organophosphorus pesticides, liquid, toxic; Organophosphorus pesticides, solid, toxic/

If material not on fire and not involved in fire: Keep sparks, flames, and other sources of ignition away. Keep material out of water sources and sewers. /Organophosphorus pesticides, solid, toxic/|If material not on fire and not involved in fire: Keep sparks, flames, and other sources of ignition away. Keep material out of water sources and sewers. Build dikes to contain flow as necessary. Attempt to stop leak if without undue personnel hazard. Use water spray to knock-down vapors. /Organophosphorus pesticides, liquid, toxic/|If material not on fire and not involved in fire: Keep sparks, flames, and other sources of ignition away. Keep material out of water sources and sewers. Build dikes to contain flow as necessary. Use water spray to knock-down vapors. /Organophosphorus pesticides, liquid, flammable, toxic/|Personnel protection: Avoid breathing dusts, and fumes from burning material. Keep upwind. Avoid bodily contact with the material. Do not handle broken packages unless wearing appropriate personal protective equipment. Wash away any material which may have contacted the body with copious amounts of water or soap and water. /Organophosphorus pesticides, solid, toxic/|For more Preventive Measures (Complete) data for EDIFENPHOS (8 total), please visit the HSDB record page.

/GUIDE 131: FLAMMABLE LIQUIDS-TOXIC/ Health: TOXIC; may be fatal if inhaled, ingested or absorbed through skin. Inhalation or contact with some of these materials will irritate or burn skin and eyes. Fire will produce irritating, corrosive and/or toxic gases. Vapors may cause dizziness or suffocation. Runoff from fire control or dilution water may cause pollution. /Organophosphorus pesticide, liquid, flammable, poisonous; Organophosphorus pesticide, liquid, flammable, toxic; Organophosphorus pesticide, liquid, poisonous, flammable; Organophosphorus pesticide, liquid, toxic, flammable/|/GUIDE 131: FLAMMABLE LIQUIDS-TOXIC/ Fire or Explosion: HIGHLY FLAMMABLE: Will be easily ignited by heat, sparks or flames. Vapors may form explosive mixtures with air. Vapors may travel to source of ignition and flash back. Most vapors are heavier than air. They will spread along ground and collect in low or confined areas (sewers, basements, tanks). Vapor explosion and poison hazard indoors, outdoors or in sewers. Those substances designated with a "P" may polymerize explosively when heated or involved in a fire. Runoff to sewer may create fire or explosion hazard. Containers may explode when heated. Many liquids are lighter than water. /Organophosphorus pesticide, liquid, flammable, poisonous; Organophosphorus pesticide, liquid, flammable, toxic; Organophosphorus pesticide, liquid, poisonous, flammable; Organophosphorus pesticide, liquid, toxic, flammable/|/GUIDE 131: FLAMMABLE LIQUIDS-TOXIC/ Public Safety: CALL Emergency Response Telephone Number ... . As an immediate precautionary measure, isolate spill or leak area for at least 50 meters (150 feet) in all directions. Keep unauthorized personnel away. Stay upwind. Keep out of low areas. Ventilate closed spaces before entering. /Organophosphorus pesticide, liquid, flammable, poisonous; Organophosphorus pesticide, liquid, flammable, toxic; Organophosphorus pesticide, liquid, poisonous, flammable; Organophosphorus pesticide, liquid, toxic, flammable/|/GUIDE 131: FLAMMABLE LIQUIDS-TOXIC/ Protective Clothing: Wear positive pressure self-contained breathing apparatus (SCBA). Wear chemical protective clothing that is specifically recommended by the manufacturer. It may provide little or no thermal protection. Structural firefighters' protective clothing provides limited protection in fire situations ONLY; it is not effective in spill situations where direct contact with the substance is possible. /Organophosphorus pesticide, liquid, flammable, poisonous; Organophosphorus pesticide, liquid, flammable, toxic; Organophosphorus pesticide, liquid, poisonous, flammable; Organophosphorus pesticide, liquid, toxic, flammable/|For more DOT Emergency Guidelines (Complete) data for EDIFENPHOS (16 total), please visit the HSDB record page.

No person may /transport,/ offer or accept a hazardous material for transportation in commerce unless that person is registered in conformance ... and the hazardous material is properly classed, described, packaged, marked, labeled, and in condition for shipment as required or authorized by ... /the hazardous materials regulations (49 CFR 171-177)./|The International Air Transport Association (IATA) Dangerous Goods Regulations are published by the IATA Dangerous Goods Board pursuant to IATA Resolutions 618 and 619 and constitute a manual of industry carrier regulations to be followed by all IATA Member airlines when transporting hazardous materials.|The International Maritime Dangerous Goods Code lays down basic principles for transporting hazardous chemicals. Detailed recommendations for individual substances and a number of recommendations for good practice are included in the classes dealing with such substances. A general index of technical names has also been compiled. This index should always be consulted when attempting to locate the appropriate procedures to be used when shipping any substance or article.|Marine Pollutant

SOIL: Edifenphos was detected in a few soil samples from 108 districts (in Korea) in year 1982(1).

highly toxic

Because different classes of enzymes may be inhibited, the effects of organophosphorus pesticide poisoning may be complex and potentially at least could involve interactions with drugs as well as with other pesticides or chemicals. Potentiation may also involve solvents or other components of formulated pesticides. Certain drugs such a phenothiazines, antihistamines, CNS depressants, barbiturates, xanthines (theophylline), aminoglycosides and parasympathomimetic agents are to be avoided because of increased toxicity. /Organophosphorus pesticides/

LD50 Rat female ip 25.5 mg/kg|LD50 Rat male ip 66.5 mg/kg|LD50 Rat oral 100 mg/kg|LC50 Rat inhalation 650 mg/cu m/ 4hr|For more Non-Human Toxicity Values (Complete) data for EDIFENPHOS (9 total), please visit the HSDB record page.

Edifenphos's former production in the US may have resulted in its release to the environment through various waste streams; it's use as a fungicide(1) results in its direct release to the environment(SRC).

TERRESTRIAL FATE: Based on a classification scheme(1), an estimated Koc value of 1,900(SRC), determined from a log Kow of 3.48(2) and a regression-derived equation(3), indicates that edifenphos is expected to have low mobility in soil(SRC). Volatilization of edifenphos from moist soil surfaces is not expected to be an important fate process(SRC) given an estimated Henry's Law constant of 7.6X10-10 atm-cu m/mole(SRC), derived from its vapor pressure, 2.7X10-7 mm Hg(4), and water solubility, 56 mg/l(5). Edifenphos is not expected to volatilize from dry soil surfaces(SRC) based upon its vapor pressure(4). During a field-study, edifenphos, applied at a rate of 5.0 mg on a paddy field soil in Japan, disappeared in 39 days with a half-life of 5-6 days(6). The percent degradation of edifenphos on a silica gel plate was greater than that observed for distilled water(7). Results suggest that edifenphos on the surface of solids such as agricultural leaves and clay particles are readily degraded as compared to natural water(7).|AQUATIC FATE: Based on a classification scheme(1), an estimated Koc value of 1,900(SRC), determined from a log Kow of 3.48(2) and a regression-derived equation(3), indicates that edifenphos is expected to adsorb to suspended solids and sediment(SRC). Volatilization from water surfaces is not expected(3) based upon an estimated Henry's Law constant of 7.6X10-10 atm-cu m/mole(SRC), derived from its vapor pressure, 2.7X10-7 mm Hg(4), and water solubility, 56 mg/l(5). According to a classification scheme(6), BCFs values ranging from 28.8 to 40.6 for fish(7) suggest bioconcentration in aquatic organisms is moderate(SRC). Edifenphos is hydrolyzed by strong acids and alkalis(5). Hydrolysis half-lives at pH 7 and 9 are 19 and 2 days, respectively(5). Based on a field study in soil(8), edifenphos is also expected to degrade in aqueous systems(SRC).|ATMOSPHERIC FATE: According to a model of gas/particle partitioning of semivolatile organic compounds in the atmosphere(1), edifenphos, which has a vapor pressure of 2.7X10-7 mm Hg at 25 °C(2), will exist in both the vapor and particulate phases in the ambient atmosphere(SRC). Vapor-phase edifenphos is degraded in the atmosphere by reaction with photochemically-produced hydroxyl radicals(SRC); the half-life for this reaction in air is estimated to be 8.7 hrs(SRC), calculated from its rate constant of 4.4X10-11 cu cm/molecule-sec at 25 °C(SRC) that was derived using a structure estimation method(3). Particulate-phase edifenphos may be removed from the air by wet and dry deposition(SRC).

The rate constant for the vapor-phase reaction of edifenphos with photochemically-produced hydroxyl radicals has been estimated as 4.4X10-11 cu cm/molecule-sec at 25 °C(SRC) using a structure estimation method(1). This corresponds to an atmospheric half-life of about 8.7 hours at an atmospheric concentration of 5X10+5 hydroxyl radicals per cu cm(1). Edifenphos is hydrolyzed by strong acids and alkalis(2). The fifty-percent disappearance times at pH 7 and 9 are 19 and 2 days, respectively(2). Edifenphos is not expected to directly photolyze due to the lack of absorption in the environmental portion of the UV spectrum (>290 nm)(SRC).

32.36|Whole fish BCFs for female guppy, male guppy, killifish, gold fish, and white cloud mountain fish are measured as 40.6, 29.3, 28.8, 38.9, and 29.3, respectively(1). According to a classification scheme(2), BCF values ranging from 28.8 to 40.6 for fish(1) suggest bioconcentration in aquatic organisms is moderate(SRC).

The Koc of edifenphos is estimated as 1,900(SRC), using a log Kow of 3.48(1) and a regression-derived equation(2). According to a classification scheme(3), this estimated Koc value suggests that edifenphos is expected to have low mobility in soil(SRC).

The Henry's Law constant for edifenphos is estimated as 7.6X10-10 atm-cu m/mole(SRC) derived from its vapor pressure, 2.7X10-7 mm Hg(1), and water solubility, 56 mg/l(2). This Henry's Law constant indicates that edifenphos is expected to be essentially nonvolatile from water and moist soil surfaces(3). The volatilization rate of edifenphos from the surface of aqueous solution was found to be 8.8X10-9 m/hr(4). Edifenphos is not expected to volatilize from dry soil surfaces(SRC) based upon its vapor pressure(1).

SURFACE WATER: Between Apr-Aug 1996, edifenphos was detected in the Shinano River, Japan at 3 sites; concns ranged from 10 to 16 ng/l (n=12)(1).|RAIN: Pesticide residues in rainwater were investigated for the estimation of pesticide flux into a lake in Ibaraki Prefecture (Japan) during the 1990 rainy season(1). The max concn of edifenphos in rainwater was estimated to be 0.36 ug/l(1).

Although no longer produced in the US, occupational exposure to edifenphos may occur through inhalation of dust and dermal contact with this compound at workplaces where edifenphos is used. (SRC)|Secondary exposure of children through contact with their parents' contaminated clothing can also occur. /Organophosphorus pesticides/

Edifenphos is rapidly absorbed following acute oral administration to rats. Within 8 hours of dosing the major part of an 35S-dose was absorbed. Within 72 hours of the administered dose, 96% in rats and > 97% in mice was excreted in the urine and feces. There were no significant sex differences noted in rats, while in mice females excreted somewhat more in the feces than corresponding males. Apart from this no sex differences in absorption and excretion were noted. Tissue residues (35S-labelled) in both rats and mice were extremely low at 72 hours. Residues in tissues following acute or sub-acute (10-daily) doses were generally low and associated with tissues concerned with distribution and excretion. Of the 6 tissues examined at various intervals after dosing (from 1 to 72 hours) the following sequence reflects tissue distribution: liver > kidney > lung > heart >> blood = brain. This same qualitative relationship of tissue distribution was reflected over the time for maximum tissue clearance.|Comparative studies on the metabolic fate of 35(S)-labeled edifenphos in rats and mice are described. Dose levels were 10 mg/kg in female rats and 20 mg/kg in mice and male rats. Only 15 to 30% of the administered radioactivity was detected in digestive organs 6 hr after administration. At 72 hr, only a trace amount of radioactivity was found over the whole body of the animals. The major part of the radioactivity was excreted via urine (75-90%) and feces (5-20%). The main metabolite of the rat is ethyl hydrogen S-phenyl phosphorothiolate (54-57%) and that of mice is dihydrogen S-phenyl phosphorothiolate (3 1-42%). Diphenyl disulfide was found in feces in both animals, but metabolites such as methyl phenyl sulfide and its derivatives were absent. Thus edifenphos is rapidly absorbed, metabolized and eliminated from mice and rats following oral administration.

The major metabolite in /the urine of/ rats was the des-S-phenyl hydrolysis product while in mice the mono-S-phenyl phosphorothioate was prevalent (further dealkylation of the major rat metabolite). In feces, small (< 2%) quantities of edifenphos were noted as well as diphenyl disulfide|...Edifenphos was degraded primarily by loss of the phenyl, thiophenyl and ethyl groups to give organophosphorus acids. The ultimate metabolites were phosphoric and sulfuric acids. Furthermore, oxidation and methylation were apparent in the later stage of metabolism, and conjugated metabolites were formed.|14C-Edifenphos was degraded in rice primarily by loss of phenyl, thiophenyl, and/or ethyl groups to give organophosphorus acids, the ultimate products being benzenesulfonic acid and presumably phosphoric acid.|Metabolism occurs principally by oxidation, hydrolysis by esterases, and by transfer of portions of the molecule to glutathione. Oxidation of organophosphorus insecticides may result in more or less toxic products. The glutathione transferase reactions produce products, that are, in most cases, of low toxicity. Hydrolytic and transferase reactions affect both thioates and their oxons. /Organophosphorus Pesticides/|For more Metabolism/Metabolites (Complete) data for EDIFENPHOS (7 total), please visit the HSDB record page.

0.09 Days

Organophosphorus insecticides exert their acute effects in both insects and mammals by inhibiting acetylcholinesterase (AChE) in the nervous system with subsequent accumulation of toxic levels of acetylcholine (ACh), which is a neurotransmitter. In many cases, the organophosphorylated enzyme is fairly stable, so that recovery from intoxication may be slow. /Organophosphorus Pesticides/

Airway protection. Ensure that a clear airway exists. Intubate the patients and aspirate the secretions with a large-bore suction device if necessary. Administer oxygen by mechanically assisted pulmonary ventilation if respiration is depressed. Improve tissue oxygenation as much as possible before administering atropine, so as to minimize the risk of ventricular fibrillation. In severe poisonings, it may be necessary to support pulmonary ventilation mechanically for several days. /Organophosphate pesticides/|Atropine sulfate. Administer atropine sulfate intravenously, or intramuscularly if intravenous injection is not possible. Remember that atropine can be administered through an endotracheal tube if initial IV access if difficult to obtain. Depending on the severity of poisoning, doses of atropine ranging from very low to...high... . The objective of atropine antidotal therapy is to antagonize the effects of excessive concentrations of acetylcholine at end-organs having muscarinic receptors. Atropine does not reactivate the cholinesterase enzyme or accelerate disposition of organophosphate. Recrudescence of poisoning may occur if tissue concentrations of organophosphate remain high when the effect of atropine wears off. Atropine is effective against muscarinic manifestations, but it is ineffective against nicotinic actions, specifically muscle weakness and twitching, and respiratory depression. Despite the limitations, atropine is often a life-saving agent in organophosphate poisonings. Favorable response to a test dose of atropine can help differentiate poisoning by anticholinesterase agents from other conditions. However, lack of response, with no evidence of atropinization (atropine refractoriness) is typical of more severe poisonings. The adjunctive use of nebulized atropine has been reported to improve respiratory distress, decrease bronchial secretions, and increase oxygenation. ...Do not administer atropine or pralidoxime prophylactically to workers exposed to organophosphate pesticides. Prophylactic dosage with either atropine or pralidoxime may mask early signs and symptoms of organophosphate poisoning and thus allow the worker to continue exposure and possibly progress to more severe poisoning. Atropine itself may enhance the health hazards of the agricultural work setting: impaired heat loss due to reduced sweating and impaired ability to operate mechanical equipment due to blurred vision. This can be caused by mydriasis, one of the effects of atropine. /Organophosphate pesticides/|Glycopyrolate has been studied as an alternative to atropine and found to have similar outcomes using continuous infusion. Ampules...of glycopyrolate were added to...saline and this infusion was titrated to the desired effects of dry mucous membranes and heart rate above 60 beats/min. During this study, atropine was used as a bolus for a heart rate less than 60 beats/min. The other apparent advantage to this regimen was a decreased number of respiratory infections. This may represent an alternative when there is a concern for respiratory infection due to excessive and difficult to control secretions, and in the presence of altered level of consciousness where the distinction between atropine toxicity or relapse of organophosphate poisoning is unclear. /Organophosphate pesticides/|Pralidoxime. Before administration of pralidoxime, draw a blood sample (heparinized) for cholinesterase analysis (since pralidoxime tends to reverse the cholinesterase depression). Administration pralidoxime (Protopam, 2-PAM), a cholinesterase reactivator, in cases of severe poisoning by organophosphate pesticides in which respiratory depression, muscle weakness, and/or twitching are severe. When administered early (usually less than 48 hours after poisoning) pralidoxime relieves the nicotinic as well as the muscarinic effects of poisoning. Pralidoxime works by reactivating the cholinesterase and also by slowing the "aging" process of phosphorylated cholinesterase to a non-reactivatable form. ... Dosage of pralidoxime may be repeated in 1-2 hours, then at 10-12 hour intervals if needed. In very severe poisonings, dosage rates may be doubled. Repeated doses of pralidoxime are usually required. In cases that involve continuing absorption of organophosphate (as after ingestion of large amounts), or continuing transfer of highly lipophilic organophosphate from fat into blood, it may be necessary to continue administration of pralidoxime for several days beyond the 48 hour post-exposure interval usually cited as the limit of its effectiveness. ... Blood pressure should be monitored during administration because of the occasional occurrence of hypertensive crisis. Administration should be slowed or stopped if blood pressure rises to hazardous levels. Be prepared to assist pulmonary ventilation mechanically if respiration is depressed during or after pralidoxime administration. If intravenous injection is not possible, pralidoxime may be given by deep intramuscular injection. /Organophosphate pesticides/|For more Antidote and Emergency Treatment (Complete) data for EDIFENPHOS (18 total), please visit the HSDB record page.

/HUMAN EXPOSURE STUDIES/ ...In human subjects sufficiently exposed to organophosphates to depress plasma- or erythrocyte-ChEs, some or all of the following behavioral variables might be impaired. In cognition: vigilance, information processing and psychomotor speed, and memory; in speech: both performance and perception; in psychic state: increased tendencies to depression, anxiety, and irritability; and in EEG records: a tendency to faster frequencies and higher voltages. /Organophosphorus Pesticides/|/HUMAN EXPOSURE STUDIES/ Respiratory and ocular symptoms are expected to appear first after exposure to airborne organophosphorus pesticides. /Organophosphorus pesticides/|/SIGNS AND SYMPTOMS/ Signs and symptoms of acute intoxication by organophosphorus insecticides include muscarinic, nicotinic, and central nervous system (CNS) manifestations. Symptoms may develop rapidly, or there may be a delay of several hours after exposure before they become evident. The delay tends to be longer in the case of more lipophilic compounds, which also require metabolic activation. Symptoms may increase in severity for more than one day and may last for several days. In severe cases, respiratory failure is a dominant effect. /Organophosphorus Pesticides/|/SIGNS AND SYMPTOMS/ The clinical picture of organophosphorus intoxication results from accumulation of ACh at nerve endings. ...The symptoms can be summarized in three groups as follows: (a) Muscarinic manifestations- increased bronchial secretion, excessive sweating, salivation, and lachrymation; pinpoint pupils, bronchoconstriction, abdominal cramps (vomiting and diarrhea); and bradycardia. (b) Nicotinic manifestations- fasciculation of fine muscles and, in more severe cases, of diaphragm and respiratory muscles; and tachycardia. (c) Central nervous system manifestations- headache, dizziness, restlessness, and anxiety; mental confusion, convulsions, and coma; and depression of the respiratory centre. All these symptoms can occur in different combinations and can vary in time of onset, sequence, and duration, depending on the chemical, dose, and route of exposure. Mild poisoning might include muscarinic and nicotinic signs only. Severe cases always show central nervous system involvement; the clinical picture is dominated by respiratory failure, sometimes leading to pulmonary edema, due to the combination of the above-mentioned symptoms. /Organophosphorus Pesticides/|For more Human Toxicity Excerpts (Complete) data for EDIFENPHOS (8 total), please visit the HSDB record page.

Bay 78418

Emulsifiable concentrate, dustable powder.|Selected tradenames: 'Hinosan' (Nihon Bayer), 'Vihino' (Vipesco) ; mixtures: 'Hinorabcide' (+ phthalide) (Nihon Bayer); 'Hinosuncide' (+ propoxur) (Nihon Bayer).

The WHO Recommended Classification of Pesticides by Hazard identifies Edifenphos (technical grade) as Class IB: highly hazardous; Main Use: fungicide, other than for seed treatment.

Product analysis by glc with FID.