Indication: 1. Congenital hypofibrinogenemia or deficiency. 2. Acquired hypofibrinogenemia: severe liver damage; liver cirrhosis; disseminated intravascular coagulation; postpartum hemorrhage and coagulation disorders caused by fibrinogen deficiency due to major surgery, trauma or internal bleeding.

Indication: 1. Congenital hypofibrinogenemia or deficiency. 2. Acquired hypofibrinogenemia: severe liver damage; liver cirrhosis; disseminated intravascular coagulation; postpartum hemorrhage and coagulation disorders caused by fibrinogen deficiency due to major surgery, trauma or internal bleeding.

Indication: 1. This product is mainly used for various bleeding caused by acute or chronic, localized or systemic primary hyperfibrinolysis. For secondary hyperfibrinolytic state caused by disseminated intravascular coagulation, this product is generally not used before heparinization; 2. It is used for traumatic or surgical bleeding in organs rich in plasminogen activators such as the prostate, urethra, lungs, brain, uterus, adrenal glands, and thyroid glands; 3. It is used as an antagonist of tissue plasminogen activator (t-PA), streptokinase, and urokinase; 4. It is used for fibrinolytic bleeding caused by artificial abortion, early placental abruption, stillbirth, and amniotic fluid embolism, as well as menorrhagia caused by pathological local increased fibrinolysis in the uterine cavity; 5. It is used for mild bleeding caused by central nervous system diseases, such as subarachnoid hemorrhage and intracranial aneurysm bleeding. The use of this product for hemostasis is superior to other anti-fibrotic drugs.

Indication: Vitamin drugs. Mainly used for coagulation disorders caused by vitamin K deficiency. For example, vitamin K deficiency caused by intestinal malabsorption. Obstructive jaundice caused by various reasons, chronic ulcerative colitis, chronic pancreatitis and reduced intestinal absorption function after extensive small bowel resection; long-term use of antibiotics can lead to vitamin K deficiency in the body, broad-spectrum antibiotics or intestinal sterilization drugs can kill or inhibit the bacterial flora in the normal intestine, resulting in a decrease in the vitamins synthesized by intestinal bacteria; the molecular structure of anticoagulants such as dicoumarol is similar to that of vitamin K, which interferes with its metabolism in the body, preventing epoxyphylloquinone from being reduced to vitamin K, and preventing vitamin K in the body from playing its role, resulting in consequences similar to vitamin K deficiency.

Indication: Vitamin drugs. Mainly used for coagulation disorders caused by vitamin K deficiency. For example, vitamin K deficiency caused by intestinal malabsorption. Obstructive jaundice caused by various reasons, chronic ulcerative colitis, chronic pancreatitis and reduced intestinal absorption function after extensive small bowel resection; long-term use of antibiotics can lead to vitamin K deficiency in the body, broad-spectrum antibiotics or intestinal sterilization drugs can kill or inhibit the bacterial flora in the normal intestine, resulting in a decrease in the vitamins synthesized by intestinal bacteria; the molecular structure of anticoagulants such as dicoumarol is similar to that of vitamin K, which interferes with its metabolism in the body, preventing epoxyphylloquinone from being reduced to vitamin K, and preventing vitamin K in the body from playing its role, resulting in consequences similar to vitamin K deficiency.