Shanxi PUDE Pharmaceutical Co., Ltd.
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Founded in:
1995-09-13 -
Country:
China -
Address:
The First Pharmaceutical Park, Datong Economic and Technological Development Zone, Shanxi Province -
Tax NO.:
91140200602167297K -
Registered Funds:
138.8 million yuan -
Website:
-
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Diammonium glycyrrhizinate |
It has certain anti-inflammatory, liver cell membrane protection and liver function improvement effects. It can reduce the increase of serum alanine aminotransferase (ALT) and aspartate aminotransferase (AST) caused by carbon tetrachloride, thioacetamide and D-aminogalactic acid, reduce the damage of D-aminogalactic acid to the liver and improve the chronic damage of immune factors to the liver.
More
It has certain anti-inflammatory, liver cell membrane protection and liver function improvement effects. It can reduce the increase of serum alanine aminotransferase (ALT) and aspartate aminotransferase (AST) caused by carbon tetrachloride, thioacetamide and D-aminogalactic acid, reduce the damage of D-aminogalactic acid to the liver and improve the chronic damage of immune factors to the liver. |
79165-06-3 | 35 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Diammonium glycyrrhizinate |
This product is a third-generation extract of the effective ingredients of Chinese medicinal liquorice, which has strong anti-inflammatory, liver cell membrane protection and liver function improvement effects. Pharmacological experiments have shown that oral administration of mice can reduce the increase of serum alanine aminotransferase and aspartate aminotransferase caused by carbon tetrachloride, thioacetamide and D-aminogalactic acid. It can also significantly reduce the morphological damage of D-aminogalactic acid to the liver and improve the chronic damage of immune factors to the liver morphology.
More
This product is a third-generation extract of the effective ingredients of Chinese medicinal liquorice, which has strong anti-inflammatory, liver cell membrane protection and liver function improvement effects. Pharmacological experiments have shown that oral administration of mice can reduce the increase of serum alanine aminotransferase and aspartate aminotransferase caused by carbon tetrachloride, thioacetamide and D-aminogalactic acid. It can also significantly reduce the morphological damage of D-aminogalactic acid to the liver and improve the chronic damage of immune factors to the liver morphology. |
79165-06-3 | 35 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Omeprazole sodium |
This product is a proton pump inhibitor of gastric parietal cells, which can specifically inhibit the secretory microtubules and H, K-ATPases on the tubular vesicles in the cytoplasm of the parietal cell apical membrane, thereby effectively inhibiting the secretion of gastric acid. It can not only non-competitively inhibit the gastric acid secretion caused by gastrin, histamine, choline, food, and stimulation of the vagus nerve, but also inhibit part of the basic gastric acid secretion that is not affected by choline or H2 receptor blockers. It also has a strong and lasting inhibitory effect on the gastric acid secretion caused by dibutyl cyclic adenosine monophosphate (DCAMP) stimulation that cannot be inhibited by H2 receptor antagonists. In addition, this product also has an inhibitory effect on the secretion of pepsin, has no obvious change in gastric mucosal blood flow, and does not affect body temperature, gastric cavity temperature, arterial blood pressure, venous hemoglobin, arterial oxygen partial pressure, carbon dioxide partial pressure and arterial blood pH.
More
This product is a proton pump inhibitor of gastric parietal cells, which can specifically inhibit the secretory microtubules and H, K-ATPases on the tubular vesicles in the cytoplasm of the parietal cell apical membrane, thereby effectively inhibiting the secretion of gastric acid. It can not only non-competitively inhibit the gastric acid secretion caused by gastrin, histamine, choline, food, and stimulation of the vagus nerve, but also inhibit part of the basic gastric acid secretion that is not affected by choline or H2 receptor blockers. It also has a strong and lasting inhibitory effect on the gastric acid secretion caused by dibutyl cyclic adenosine monophosphate (DCAMP) stimulation that cannot be inhibited by H2 receptor antagonists. In addition, this product also has an inhibitory effect on the secretion of pepsin, has no obvious change in gastric mucosal blood flow, and does not affect body temperature, gastric cavity temperature, arterial blood pressure, venous hemoglobin, arterial oxygen partial pressure, carbon dioxide partial pressure and arterial blood pH. |
95510-70-6 | 25 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| L-Lysine hydrochloride |
L-lysine is one of the eight essential amino acids for the human body. It can promote human development, enhance immune function, and improve the function of central nervous tissue.
More
L-lysine is one of the eight essential amino acids for the human body. It can promote human development, enhance immune function, and improve the function of central nervous tissue. |
10098-89-2 | 16 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| L-Lysine hydrochloride |
L-lysine is one of the eight essential amino acids for the human body. It can promote human development, enhance immune function, and improve the function of central nervous tissue.
More
L-lysine is one of the eight essential amino acids for the human body. It can promote human development, enhance immune function, and improve the function of central nervous tissue. |
10098-89-2 | 16 |