This product is a blood lipid regulating drug of clofibric acid derivatives. It may reduce the cAMP content in liver microsomes, increase the activity of lipoprotein esterase, and decompose lipids in lipoproteins. Experimental and clinical studies have shown that this product reduces blood cholesterol and triglycerides and increases blood high-density lipoprotein. In addition, this product also has anti-platelet aggregation and anti-thrombotic effects and reduces blood uric acid.

This product is a fluoroquinolone antibacterial drug with a broad-spectrum antibacterial effect, especially high antibacterial activity against aerobic Gram-negative bacteria. It has good antibacterial activity against the following bacteria in vitro: most bacteria of the Enterobacteriaceae family, including Citrobacter, Enterobacter cloacae, Enterobacter aerogenes and other Enterobacter species, Escherichia coli, Klebsiella, Proteus, Salmonella, Shigella, Vibrio, Yersinia, etc. Norfloxacin also has antibacterial activity against multi-drug resistant bacteria in vitro. It also has good antibacterial effects on penicillin-resistant Neisseria gonorrhoeae, Haemophilus influenzae and Moraxella catarrhalis. Norfloxacin is a bactericide that acts on the A subunit of bacterial DNA helicase, inhibiting DNA synthesis and replication, leading to bacterial death.

1. Effects on water and electrolyte excretion: diuretic effect, increased excretion of urinary sodium, potassium, chloride, phosphorus and magnesium, and decreased excretion of urinary calcium. By inhibiting the reabsorption of sodium chloride in the distal tubule anterior segment and proximal tubule, increasing Na-K exchange in the distal tubule and collecting duct, K secretion increases. It may also inhibit carbonic anhydrase activity and phosphodiesterase activity, reduce the uptake of fatty acids and mitochondrial oxygen consumption by the renal tubule, thereby inhibiting the active reabsorption of Na and Cl- by the renal tubule. 2. Antihypertensive effect: In addition to diuresis and sodium excretion, there may be extrarenal mechanisms involved in blood pressure reduction, such as increased gastrointestinal excretion of Na. 3. Effects on renal hemodynamics and glomerular filtration function: Due to the reduced reabsorption of water and Na by the renal tubule, the pressure in the renal tubule increases, stimulating the macula densa through the tubule-glomerular reflex, increasing the secretion of renin and angiotensin in the kidney, causing renal vasoconstriction, decreased renal blood flow, contraction of the glomerular afferent and efferent arterioles, and decreased glomerular filtration rate.

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It works by acting on the 50S subunit of bacterial ribosomes, inhibiting the synthesis of bacterial proteins, and is a growth-stage antibacterial drug. Its antibacterial properties are similar to those of erythromycin, showing strong antibacterial effects on Gram-positive bacteria and mycoplasmas, and also on some Gram-negative bacteria such as Neisseria meningitidis and Neisseria gonorrhoeae.