Toxic symptoms after taking an overdose of cefaclor include nausea, vomiting, upper abdominal discomfort and diarrhea, the severity of which is dose-related. If other symptoms are present, they may be secondary to the original disease, allergic reaction or other toxic effects. When treating drug overdose, the possibility of multiple drug overdose, drug interactions and individual differences in patient pharmacokinetics should be considered. Unless five times the normal dose of cefaclor is taken, it is not necessary to clear the excess drug from the gastrointestinal tract. Pay attention to protecting the patient's airway and maintaining ventilation and perfusion. Closely monitor and maintain the patient's vital signs, blood gas and serum electrolytes within an acceptable range. Giving activated carbon can reduce the absorption of drugs through the gastrointestinal tract. Activated carbon is more effective than vomiting and lavage in many cases. Consider using activated carbon instead of gastric emptying, or in addition to gastric emptying. Multiple administrations of activated carbon may accelerate the elimination of some absorbed drugs. When performing gastric emptying or giving activated carbon, pay attention to protecting the patient's airway. Forced catheterization, peritoneal dialysis, hemodialysis, or carbon hemoperfusion have not been shown to be beneficial in the setting of cefaclor overdose.

Toxic symptoms after taking an overdose of cefaclor include nausea, vomiting, upper abdominal discomfort and diarrhea, the severity of which is dose-related. If other symptoms are present, they may be secondary to the original disease, allergic reaction or other toxic effects.

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This product is a broad-spectrum antibiotic, which has antibacterial activity against some Gram-positive and Gram-negative bacteria, especially against Streptococcus (except Enterococcus) and Pneumococcus among Gram-positive bacteria, and gonococci, Branhamella, Escherichia coli, Klebsiella, Serratia, Proteus, and influenza bacilli among Gram-negative bacteria. Its mechanism of action is bactericidal. This product has extremely strong stability against β-lactamase produced by various bacteria, and shows excellent antibacterial activity against bacteria producing β-lactamase. The mechanism of action of this product is to prevent the synthesis of bacterial cell walls. Its action point varies according to the strain, and it has a high affinity with 1 (la, lb, 1c) and 3 in penicillin binding protein (PBP).