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Founded in:
1998-11-03 -
Country:
China -
Address:
Area A, 4th Floor, Digital Peninsula, No. 289, Hongliu Road, Fubao Community, Fubao Street, Futian District, Shenzhen -
Tax NO.:
91440300708453259J -
Registered Funds:
1,114.816535 yuan -
Website:
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Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Cefoxitin sodium |
It kills bacteria by inhibiting bacterial cell wall synthesis and has high resistance to β-lactamase produced by bacteria. Some common Gram-positive, Gram-negative aerobic and anaerobic pathogens are highly sensitive to this product. It is resistant to Pseudomonas aeruginosa, most strains of enterococci, and Escherichia coli.
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It kills bacteria by inhibiting bacterial cell wall synthesis and has high resistance to β-lactamase produced by bacteria. Some common Gram-positive, Gram-negative aerobic and anaerobic pathogens are highly sensitive to this product. It is resistant to Pseudomonas aeruginosa, most strains of enterococci, and Escherichia coli. |
33564-30-6 | 23 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Cefoxitin sodium |
It kills bacteria by inhibiting bacterial cell wall synthesis and has high resistance to β-lactamase produced by bacteria. Some common Gram-positive, Gram-negative aerobic and anaerobic pathogens are highly sensitive to this product. It is resistant to Pseudomonas aeruginosa, most strains of enterococci, and Escherichia coli.
More
It kills bacteria by inhibiting bacterial cell wall synthesis and has high resistance to β-lactamase produced by bacteria. Some common Gram-positive, Gram-negative aerobic and anaerobic pathogens are highly sensitive to this product. It is resistant to Pseudomonas aeruginosa, most strains of enterococci, and Escherichia coli. |
33564-30-6 | 23 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Levetiracetam |
This product is a pyrrolidone derivative, and its chemical structure is different from that of traditional anti-epileptic drugs. Its mechanism of action is still unclear. In vivo and in vitro tests have shown that this product does not change cell characteristics and neurotransmission function. Animal experiments have confirmed that this product is effective for partial epileptic seizures and generalized seizures without convulsions; in human clinical applications, it has also been confirmed that this product is effective for partial and generalized epileptic seizures. The effective dose and toxic dose of this product are far apart, and the safety is relatively good.
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This product is a pyrrolidone derivative, and its chemical structure is different from that of traditional anti-epileptic drugs. Its mechanism of action is still unclear. In vivo and in vitro tests have shown that this product does not change cell characteristics and neurotransmission function. Animal experiments have confirmed that this product is effective for partial epileptic seizures and generalized seizures without convulsions; in human clinical applications, it has also been confirmed that this product is effective for partial and generalized epileptic seizures. The effective dose and toxic dose of this product are far apart, and the safety is relatively good. |
102767-28-2 | 93 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| TICAGRELOR |
|
274693-27-5 | 93 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Benazepril hydrochloride |
This product is hydrolyzed into benazeprilat in the liver, becoming a competitive angiotensin converting enzyme inhibitor, preventing angiotensin I from converting to angiotensin II, reducing vascular resistance, reducing aldosterone secretion, and increasing plasma renin activity. Benazeprilat also inhibits the degradation of bradykinin, which also reduces vascular resistance and produces a hypotensive effect. In addition, this product dilates arteries and veins, reduces peripheral vascular resistance or cardiac afterload, reduces pulmonary capillary entrapment pressure or cardiac preload, and also reduces pulmonary vascular resistance, thereby improving cardiac output and prolonging exercise tolerance and time.
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This product is hydrolyzed into benazeprilat in the liver, becoming a competitive angiotensin converting enzyme inhibitor, preventing angiotensin I from converting to angiotensin II, reducing vascular resistance, reducing aldosterone secretion, and increasing plasma renin activity. Benazeprilat also inhibits the degradation of bradykinin, which also reduces vascular resistance and produces a hypotensive effect. In addition, this product dilates arteries and veins, reduces peripheral vascular resistance or cardiac afterload, reduces pulmonary capillary entrapment pressure or cardiac preload, and also reduces pulmonary vascular resistance, thereby improving cardiac output and prolonging exercise tolerance and time. |
86541-74-4 | 31 |