This product is a new type of non-narcotic analgesic, with mild antipyretic and muscle relaxant effects. Its chemical structure belongs to cyclized o-methylbenzylamine. Therefore, it does not have the characteristics of non-steroidal anti-inflammatory drugs, nor is it an opioid receptor agonist. It is effective for moderate and severe pain. An intramuscular injection of 20 mg of this product is equivalent to the effect of 12 mg of morphine. It has a mild respiratory depressant effect. It has no depressant effect on the circulatory system. There is no tolerance and dependence.

In severe hepatitis or liver dysfunction, the liver's conversion of ammonia to urea is impaired, resulting in increased blood ammonia and encephalopathy symptoms. The intake of glutamate and arginine is beneficial to reduce and eliminate blood ammonia, thereby improving encephalopathy symptoms.

1. Effects on water and electrolyte excretion: diuretic effect, increased excretion of urinary sodium, potassium, chloride, phosphorus and magnesium, and decreased excretion of urinary calcium; inhibiting the reabsorption of sodium chloride in the distal tubule proximal and proximal tubules, increasing Na-K exchange in the distal tubules and collecting ducts, and increasing K secretion. It may work by inhibiting carbonic anhydrase activity and phosphodiesterase activity. 2. Antihypertensive effect: In addition to diuresis and sodium excretion, there may be extrarenal mechanisms involved, such as increased gastrointestinal excretion of Na. 3. Effects on renal hemodynamics and glomerular filtration function: reduced reabsorption of water and Na by the renal tubules, increased pressure in the renal tubules, increased water and Na flowing through the distal convoluted tubules, stimulating the macula densa through the tubule-glomerular reflex, increasing the secretion of renin and angiotensin in the kidney, causing renal vasoconstriction, decreased renal blood flow, contraction of the glomerular afferent and efferent arterioles, and decreased glomerular filtration rate.

This product mainly acts on α receptors, directly stimulating α receptors. Its effect is weaker than that of norepinephrine but more persistent. Its cardiovascular effect is similar to that of norepinephrine. It can constrict blood vessels, continuously increase systolic and diastolic blood pressure, and enhance myocardial contractility. The cardiac output of normal people does not change much, but it can increase the cardiac output of patients with shock. The excitement of heart rate is not very significant, rarely causes arrhythmia, and has no central nervous system excitation effect. Because its pressor effect is reliable and lasts for a long time, it rarely causes reactions such as palpitations or decreased urine volume. When administered continuously, because this product indirectly replaces the transmitter in the adrenergic nerve vesicles, it can reduce the transmitter and weaken the intrinsic effect. Therefore, it cannot be stopped suddenly to avoid rebound hypotension.