Can neutralize excess stomach acid

Can neutralize excess stomach acid

It can not only inhibit gastric juice secretion, relieve gastric smooth muscle spasm, but also delay gastric emptying.

It is a local irritant that can selectively act on the cold receptors of the mucosa, produce a cold reflex, cause the mucosal blood vessels to contract, and produce a therapeutic effect. It has a cooling effect on the mucosa; it can cause blood vessels to contract and reduce edema when used on inflamed mucosa.

The active ingredient of this product, menthol, is a local irritant. When used locally, it can selectively act on the cold receptors of the mucosa, produce a cold reflex, cause the mucosal blood vessels to contract, and produce a therapeutic effect. When used on the mucosa, it has a cooling effect; when used on inflamed mucosa, it can cause blood vessels to contract and reduce edema.

The active ingredient of this product, menthol, is a local irritant. When used locally, it can selectively act on the cold receptors of the mucosa, produce a cold reflex, cause the mucosal blood vessels to contract, and produce a therapeutic effect. When used on the mucosa, it has a cooling effect; when used on inflamed mucosa, it can cause blood vessels to contract and reduce edema.

1. It is a non-selective competitive inhibitor of adrenergic β-receptor blocker, blocking β1 and β2 receptors on the heart, reducing the contractile force and contractile speed of the heart, while inhibiting the contraction of vascular smooth muscle, reducing myocardial oxygen consumption, and is used to treat angina pectoris; 2. It inhibits the adrenergic excitement of cardiac pacemaker potentials and is used to treat arrhythmias; 3. It can inhibit the release of renin and reduce cardiac output through central and adrenergic neuron blocking, and is used to treat hypertension; 4. It competitively antagonizes the effects of isoproterenol and norepinephrine, blocks β2 receptors, reduces plasma renin activity, and has a significant anti-platelet aggregation effect.

It is a coenzyme that improves the body's metabolism and participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotides in the body. It is also the main source of energy in the body. When energy is needed for absorption, secretion, muscle contraction and biochemical synthesis reactions, adenosine triphosphate is decomposed into adenosine diphosphate and phosphate groups, and energy is released at the same time. Animal experiments can inhibit the slow calcium ion influx of slow-reacting fibers, block or delay the forward conduction in the atrioventricular node reentry pathway, and large doses may also block or delay the forward and reverse conduction of the bypass pathway; in addition, it also has a short-term and strong effect of enhancing the vagus nerve, thereby terminating arrhythmias caused by atrioventricular node reentry and bypass reentry mechanisms.

It is a coenzyme that improves the body's metabolism and participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotides in the body. It is also the main source of energy in the body. When energy is needed for absorption, secretion, muscle contraction and biochemical synthesis reactions, adenosine triphosphate is decomposed into adenosine diphosphate and phosphate groups, and energy is released at the same time. Animal experiments can inhibit the slow calcium ion influx of slow-reacting fibers, block or delay the forward conduction in the atrioventricular node reentry pathway, and large doses may also block or delay the forward and reverse conduction of the bypass pathway; in addition, it also has a short-term and strong effect of enhancing the vagus nerve, thereby terminating arrhythmias caused by atrioventricular node reentry and bypass reentry mechanisms.