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Founded in:
2001-08-06 -
Country:
China -
Address:
No. 1 Ningbo Road, Shuyang Economic and Technological Development Zone, Jiangsu Province -
Tax NO.:
9132132273013991X6 -
Registered Funds:
45 million yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Trimethoprim |
It selectively inhibits the action of dihydrofolate reductase. When used in combination with sulfamethoxazole, it can double-block the bacterial folate metabolism, enhance the synergistic antibacterial effect, and reduce drug-resistant strains.
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It selectively inhibits the action of dihydrofolate reductase. When used in combination with sulfamethoxazole, it can double-block the bacterial folate metabolism, enhance the synergistic antibacterial effect, and reduce drug-resistant strains. |
738-70-5 | 44 | |
| Sulfamethoxazole |
It acts on dihydrofolate synthase, interfering with the first step of folic acid synthesis. When used in combination with trimethoprim, it can doubly block the bacterial folic acid metabolism, enhance the synergistic antibacterial effect, and reduce drug-resistant strains.
More
It acts on dihydrofolate synthase, interfering with the first step of folic acid synthesis. When used in combination with trimethoprim, it can doubly block the bacterial folic acid metabolism, enhance the synergistic antibacterial effect, and reduce drug-resistant strains. |
723-46-6 | 33 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Calcium folinatc |
This product is a formyl derivative of tetrahydrofolate, mainly used for the rescue of high-dose methotrexate and other folic acid antagonists. It is converted into tetrahydrofolate by tetrahydrofolate reductase, which can effectively counteract the toxic reaction caused by methotrexate, but has no obvious effect on the existing methotrexate neurotoxicity.
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This product is a formyl derivative of tetrahydrofolate, mainly used for the rescue of high-dose methotrexate and other folic acid antagonists. It is converted into tetrahydrofolate by tetrahydrofolate reductase, which can effectively counteract the toxic reaction caused by methotrexate, but has no obvious effect on the existing methotrexate neurotoxicity. |
6035-45-6 | 6 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Calcium folinatc |
This product is a formyl derivative of tetrahydrofolate, mainly used for the rescue of high-dose methotrexate and other folic acid antagonists. It is converted into tetrahydrofolate by tetrahydrofolate reductase, which can effectively counteract the toxic reaction caused by methotrexate, but has no obvious effect on the existing methotrexate neurotoxicity.
More
This product is a formyl derivative of tetrahydrofolate, mainly used for the rescue of high-dose methotrexate and other folic acid antagonists. It is converted into tetrahydrofolate by tetrahydrofolate reductase, which can effectively counteract the toxic reaction caused by methotrexate, but has no obvious effect on the existing methotrexate neurotoxicity. |
6035-45-6 | 6 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Pentoxifylline |
It improves blood rheology by reducing blood viscosity, reduces blood viscosity in a dose-dependent manner, improves red blood cell deformability, improves the blood rheological properties of white blood cells, and inhibits the adhesion and activation of neutrophils. For patients with chronic peripheral arterial vascular disease, it can increase blood flow, affect microcirculation, and improve tissue oxygen supply.
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It improves blood rheology by reducing blood viscosity, reduces blood viscosity in a dose-dependent manner, improves red blood cell deformability, improves the blood rheological properties of white blood cells, and inhibits the adhesion and activation of neutrophils. For patients with chronic peripheral arterial vascular disease, it can increase blood flow, affect microcirculation, and improve tissue oxygen supply. |
6493-05-6 | 42 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Tiopronin |
Animal experiments have shown that this product can reduce the increase of serum AST and ALT in animal acute liver injury models caused by thioacetamide and carbon tetrachloride, and inhibit the accumulation of triglycerides caused by chronic liver injury models; it can promote liver glycogen synthesis, inhibit the increase of cholesterol, and help to restore the serum albumin/globulin ratio.
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Animal experiments have shown that this product can reduce the increase of serum AST and ALT in animal acute liver injury models caused by thioacetamide and carbon tetrachloride, and inhibit the accumulation of triglycerides caused by chronic liver injury models; it can promote liver glycogen synthesis, inhibit the increase of cholesterol, and help to restore the serum albumin/globulin ratio. |
1953-02-2 | 55 |