By protecting the energy metabolism of cells under hypoxia or ischemia, preventing the decrease of intracellular ATP levels, it ensures the normal function of ion pumps and the normal operation of transmembrane sodium-potassium flow, and maintains the stability of the intracellular environment. It helps maintain the energy metabolism of the heart and nerve sensory organs under ischemia and hypoxia, reduces intracellular acidosis and changes in transmembrane ion flow caused by ischemia, reduces the movement and infiltration of multinuclear neutrophils during ischemia and myocardial reperfusion, and reduces the scope of experimental infarction. While producing this effect, it has no significant effect on hemodynamics. It increases coronary blood flow reserve, delays the occurrence of exercise-induced ischemia, limits rapid fluctuations in blood pressure without significant changes in heart rate, significantly reduces the frequency of angina attacks, and significantly reduces the use of glyceryl trinitrate.

It is a potent and highly selective competitive antagonist of 5-hydroxytryptamine 3 (5-HT3) receptors in peripheral neurons and the central nervous system. It inhibits the vomiting reflex mainly by selectively blocking the presynaptic 5-HT3 receptors of peripheral neurons. In addition, its antiemetic effect may also be related to its inhibition of the stimulation of the vagus nerve in the rear region by directly blocking the central 5-HT3 receptors.

It has a significant bactericidal effect on Mycobacterium tuberculosis and some non-tuberculous mycobacteria (including Mycobacterium leprae, etc.) both inside and outside the host cells.

Anti-tuberculosis drugs are a combination of rifampicin, isoniazid and pyrazinamide. Rifampicin has a significant bactericidal effect on Mycobacterium tuberculosis and some non-tuberculous mycobacteria (including Mycobacterium leprae) both inside and outside the host cells.

Anti-tuberculosis drugs are a combination of rifampicin, isoniazid and pyrazinamide. Rifampicin has a significant bactericidal effect on Mycobacterium tuberculosis and some non-tuberculous mycobacteria (including Mycobacterium leprae) both inside and outside the host cells.

This product is a coenzyme. Adenosine triphosphate disodium is a nucleotide derivative that participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotide in the body. When energy is needed for absorption, secretion, muscle contraction and biochemical synthesis reactions in the body, adenosine triphosphate is decomposed into adenosine diphosphate and phosphate groups, and energy is released at the same time. Adenosine triphosphate disodium can penetrate the blood-cerebrospinal fluid barrier, improve the stability and reconstruction ability of the membrane structure of nerve cells, and promote the regrowth of neurites.