Antiepileptic drugs, antiarrhythmic drugs. The therapeutic dose does not cause sedation and hypnosis, and has a selective antagonistic effect on the tonic phase of super-strong electric shock and convulsion, but is ineffective or even aggravates the spasm phase. Therefore, it is effective for grand mal epileptic seizures, but ineffective for absence seizures. Its antiepileptic mechanism is generally believed to increase the outflow of sodium ions from cells, reduce the inflow of sodium ions, stabilize the nerve cell membrane, increase the excitation threshold, and reduce the spread of high-frequency discharges in the lesion. In addition, this product shortens the action potential interval and the effective refractory period, and can also inhibit the influx of calcium ions, reduce myocardial autonomy, inhibit the sympathetic center, and have an inhibitory effect on the ectopic rhythm points of the atria and ventricles, and increase the thresholds for atrial fibrillation and ventricular fibrillation.

It achieves bactericidal effect by inhibiting the synthesis of bacterial cell wall. It has bactericidal effect on a wide range of common clinical bacteria and can resist hydrolysis by multiple beta-lactamases. Bacteria sensitive to this product include Gram-positive bacteria and Gram-negative bacteria, as well as some anaerobic bacteria.

This product is a third-generation cephalosporin antibiotic. It has high antibacterial activity against Escherichia coli, Klebsiella pneumoniae and other Enterobacteriaceae, Haemophilus influenzae, Pseudomonas aeruginosa, etc. It also has good antibacterial effects on nitrate-negative bacilli, Alcaligenes, etc. Most beta-lactamases produced by bacteria are highly stable, so it can still have antibacterial activity against multi-drug resistant strains of most of the above-mentioned Gram-negative bacilli. Gram-positive cocci such as pneumococci and hemolytic streptococci are highly sensitive to this product, but this product has only moderate activity against staphylococci, while enterococci and methicillin-resistant Staphylococci are often resistant to this product. This product has certain antibacterial activity against anaerobic bacteria such as Peptococcus and Peptostreptococci, but has poor antibacterial effect against Bacteroides fragilis. Its mechanism of action is to bind to penicillin-binding proteins (PBPs) on the bacterial cell membrane, acylate the transpeptidase, inhibit the synthesis of the bacterial septum and cell wall, affect the cross-linking of the cell wall mucopeptide components, inhibit cell division and growth, elongate the bacterial morphology, and finally dissolve and die.

Ganciclovir belongs to the guanine antiviral drugs. It is a homologue of acyclovir but has a stronger effect. It has a strong inhibitory effect on cytomegalovirus in AIDS patients. After entering the cell, it is quickly phosphorylated to form a triphosphate compound. The phosphorylation process is faster in cells infected with cytomegalovirus. Its triphosphate can competitively inhibit DNA polymerase and be incorporated into the DNA of viruses and host cells, thereby inhibiting DNA synthesis. The inhibitory effect on viral DNA polymerase is stronger than that on host cell DNA polymerase.

Ganciclovir is a guanine antiviral drug. It is a homologue of acyclovir, but has a stronger effect, especially on cytomegalovirus in AIDS patients. After entering the cell, this product is rapidly phosphorylated to form a monophosphate compound, and then converted into a triphosphate compound by the action of cellular kinase. In cells infected with cytomegalovirus, its phosphorylation process is faster than that in normal cells. The triphosphate of this drug can competitively inhibit DNA polymerase and be incorporated into the DNA of viruses and host cells, thereby inhibiting DNA synthesis. This drug has a stronger inhibitory effect on viral DNA polymerase than on host cell DNA polymerase.