Xinrong improves myocardial ischemia-hypoxic necrosis in rats induced by isoproterenol, reduces myocardial oxygen consumption, and alleviates myocardial necrosis; it has a protective effect on myocardial cell damage in rats and neonates; it improves the electrocardiogram of myocardial obstructive ischemia injury in rats after coronary artery ligation; it reduces the TBA value in plasma and resists lipid peroxidation; it reduces the activity of enzymes such as hydroxybutyrate dehydrogenase (CPK) in plasma; it reduces pulmonary artery pressure (MPAP) and pulmonary wedge pressure (PCWP), and increases the cardiac index (C1); it increases the activity of erythrocyte superoxide dismutase (SOD), and reduces the content of lipid peroxide (LPO) in plasma.

Xinrong improves myocardial ischemia-hypoxic necrosis in rats induced by isoproterenol, reduces myocardial oxygen consumption, and alleviates myocardial necrosis; it has a protective effect on myocardial cell damage in rats and neonates; it improves the electrocardiogram of myocardial obstructive ischemia injury in rats after coronary artery ligation; it reduces the TBA value in plasma and resists lipid peroxidation; it reduces the activity of enzymes such as hydroxybutyrate dehydrogenase (CPK) in plasma; it reduces pulmonary artery pressure (MPAP) and pulmonary wedge pressure (PCWP), and increases the cardiac index (C1); it increases the activity of erythrocyte superoxide dismutase (SOD), and reduces the content of lipid peroxide (LPO) in plasma.

Xinrong improves myocardial ischemia-hypoxic necrosis in rats induced by isoproterenol, reduces myocardial oxygen consumption, and alleviates myocardial necrosis; it has a protective effect on myocardial cell damage in rats and neonates; it improves the electrocardiogram of myocardial obstructive ischemia injury in rats after coronary artery ligation; it reduces the TBA value in plasma and resists lipid peroxidation; it reduces the activity of enzymes such as hydroxybutyrate dehydrogenase (CPK) in plasma; it reduces pulmonary artery pressure (MPAP) and pulmonary wedge pressure (PCWP), and increases the cardiac index (C1); it increases the activity of erythrocyte superoxide dismutase (SOD), and reduces the content of lipid peroxide (LPO) in plasma.

Xinrong improves myocardial ischemia-hypoxic necrosis in rats induced by isoproterenol, reduces myocardial oxygen consumption, and alleviates myocardial necrosis; it has a protective effect on myocardial cell damage in rats and neonates; it improves the electrocardiogram of myocardial obstructive ischemia injury in rats after coronary artery ligation; it reduces the TBA value in plasma and resists lipid peroxidation; it reduces the activity of enzymes such as hydroxybutyrate dehydrogenase (CPK) in plasma; it reduces pulmonary artery pressure (MPAP) and pulmonary wedge pressure (PCWP), and increases the cardiac index (C1); it increases the activity of erythrocyte superoxide dismutase (SOD), and reduces the content of lipid peroxide (LPO) in plasma.

Xinrong improves myocardial ischemia-hypoxic necrosis in rats induced by isoproterenol, reduces myocardial oxygen consumption, and alleviates myocardial necrosis; it has a protective effect on myocardial cell damage in rats and neonates; it improves the electrocardiogram of myocardial obstructive ischemia injury in rats after coronary artery ligation; it reduces the TBA value in plasma and resists lipid peroxidation; it reduces the activity of enzymes such as hydroxybutyrate dehydrogenase (CPK) in plasma; it reduces pulmonary artery pressure (MPAP) and pulmonary wedge pressure (PCWP), and increases the cardiac index (C1); it increases the activity of erythrocyte superoxide dismutase (SOD), and reduces the content of lipid peroxide (LPO) in plasma.

Xinrong improves myocardial ischemia-hypoxic necrosis in rats induced by isoproterenol, reduces myocardial oxygen consumption, and alleviates myocardial necrosis; it has a protective effect on myocardial cell damage in rats and neonates; it improves the electrocardiogram of myocardial obstructive ischemia injury in rats after coronary artery ligation; it reduces the TBA value in plasma and resists lipid peroxidation; it reduces the activity of enzymes such as hydroxybutyrate dehydrogenase (CPK) in plasma; it reduces pulmonary artery pressure (MPAP) and pulmonary wedge pressure (PCWP), and increases the cardiac index (C1); it increases the activity of erythrocyte superoxide dismutase (SOD), and reduces the content of lipid peroxide (LPO) in plasma.

Improve myocardial ischemia and hypoxia, reduce myocardial oxygen consumption, and alleviate myocardial necrosis; have a protective effect on myocardial cell damage; reduce pulmonary artery pressure and pulmonary wedge pressure, and improve cardiac index; increase erythrocyte superoxide dismutase activity and reduce lipid peroxide content.

Improve myocardial ischemia and hypoxia, reduce myocardial oxygen consumption, and alleviate myocardial necrosis; have a protective effect on myocardial cell damage; reduce pulmonary artery pressure and pulmonary wedge pressure, and improve cardiac index; increase erythrocyte superoxide dismutase activity and reduce lipid peroxide content.

Improve myocardial ischemia and hypoxia, reduce myocardial oxygen consumption, and alleviate myocardial necrosis; have a protective effect on myocardial cell damage; reduce pulmonary artery pressure and pulmonary wedge pressure, and improve cardiac index; increase erythrocyte superoxide dismutase activity and reduce lipid peroxide content.

Improve myocardial ischemia and hypoxia, reduce myocardial oxygen consumption, and alleviate myocardial necrosis; have a protective effect on myocardial cell damage; resist lipid peroxidation, reduce the activity of hydroxybutyrate dehydrogenase and creatine phosphokinase in plasma; reduce pulmonary artery pressure and pulmonary wedge pressure, and improve cardiac index; increase erythrocyte superoxide dismutase activity and reduce lipid peroxide content.

Improve myocardial ischemia and hypoxia, reduce myocardial oxygen consumption, and alleviate myocardial necrosis; have a protective effect on myocardial cell damage; reduce pulmonary artery pressure and pulmonary wedge pressure, and improve cardiac index; increase erythrocyte superoxide dismutase activity and reduce lipid peroxide content.

Improve myocardial ischemia and hypoxia, reduce myocardial oxygen consumption, and alleviate myocardial necrosis; have a protective effect on myocardial cell damage; reduce pulmonary artery pressure and pulmonary wedge pressure, and improve cardiac index; increase erythrocyte superoxide dismutase activity and reduce lipid peroxide content.

No specific pharmacological effects mentioned