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Founded in:
2000-01-14 -
Country:
China -
Address:
Yingshang Industrial Development Zone, Fuyang City, Anhui Province -
Tax NO.:
91341226738907020P -
Registered Funds:
30 million yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Naproxen |
This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins.
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This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins. |
0.1g | 22204-53-1 | 30 |
| Starch |
This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins.
More
This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins. |
9005-25-8 | 79 | |
| Hydroxypropyl methyl cellulose |
This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins.
More
This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins. |
9004-65-3 | 56 | |
| Magnesium stearate |
This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins.
More
This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins. |
557-04-0 | 61 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Pipemidic acid |
Quinolone antibiotics interfere with bacterial DNA synthesis by acting on bacterial DNA gyrase, thus causing bacterial death. They have antibacterial effects on Gram-negative bacteria such as Escherichia coli, Klebsiella pneumoniae, Enterobacter aerogenes, Proteus mirabilis, Serratia, Salmonella typhi, Shigella, Pseudomonas aeruginosa, etc.
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Quinolone antibiotics interfere with bacterial DNA synthesis by acting on bacterial DNA gyrase, thus causing bacterial death. They have antibacterial effects on Gram-negative bacteria such as Escherichia coli, Klebsiella pneumoniae, Enterobacter aerogenes, Proteus mirabilis, Serratia, Salmonella typhi, Shigella, Pseudomonas aeruginosa, etc. |
51940-44-4 | 3 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Pipemidic acid |
Quinolone antibiotics interfere with bacterial DNA synthesis by acting on bacterial DNA gyrase, thus causing bacterial death. They have antibacterial effects on Gram-negative bacteria such as Escherichia coli, Klebsiella pneumoniae, Enterobacter aerogenes, Proteus mirabilis, Serratia, Salmonella typhi, Shigella, Pseudomonas aeruginosa, etc.
More
Quinolone antibiotics interfere with bacterial DNA synthesis by acting on bacterial DNA gyrase, thus causing bacterial death. They have antibacterial effects on Gram-negative bacteria such as Escherichia coli, Klebsiella pneumoniae, Enterobacter aerogenes, Proteus mirabilis, Serratia, Salmonella typhi, Shigella, Pseudomonas aeruginosa, etc. |
51940-44-4 | 3 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Aceglutamide |
After passing through the blood-cerebrospinal fluid barrier, it is decomposed into glutamate and gamma-aminobutyric acid (GABA). Glutamate is involved in the information transmission of the central nervous system. Gamma-aminobutyric acid can antagonize the excitotoxic effects of glutamate, improve nerve cell metabolism, maintain nerve stress resistance, reduce the effect of blood ammonia, and improve brain function.
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After passing through the blood-cerebrospinal fluid barrier, it is decomposed into glutamate and gamma-aminobutyric acid (GABA). Glutamate is involved in the information transmission of the central nervous system. Gamma-aminobutyric acid can antagonize the excitotoxic effects of glutamate, improve nerve cell metabolism, maintain nerve stress resistance, reduce the effect of blood ammonia, and improve brain function. |
2490-97-3 | 6 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| 16α-Methyl-11β,17α,21-trihydroxy-9α-fluoropregnan-1,4-diene-3,20-dione-21-acetate |
Adrenal cortical hormone drugs have more significant anti-inflammatory, anti-allergic, and anti-shock effects than prednisone, but have little effect on water and sodium retention and promoting potassium excretion, and have a stronger inhibitory effect on the pituitary-adrenal gland. 1 Anti-inflammatory effect: This product can reduce and prevent tissue response to inflammation, thereby reducing the manifestations of inflammation. Hormones inhibit the accumulation of inflammatory cells, including macrophages and white blood cells at the site of inflammation, and inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. It can reduce and prevent tissue response to inflammation, thereby reducing the manifestations of inflammation. 2 Immunosuppressive effect: including preventing or inhibiting cell-mediated immune responses, delayed allergic reactions, reducing T lymphocytes, monocytes, and eosinophils
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Adrenal cortical hormone drugs have more significant anti-inflammatory, anti-allergic, and anti-shock effects than prednisone, but have little effect on water and sodium retention and promoting potassium excretion, and have a stronger inhibitory effect on the pituitary-adrenal gland. 1 Anti-inflammatory effect: This product can reduce and prevent tissue response to inflammation, thereby reducing the manifestations of inflammation. Hormones inhibit the accumulation of inflammatory cells, including macrophages and white blood cells at the site of inflammation, and inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. It can reduce and prevent tissue response to inflammation, thereby reducing the manifestations of inflammation. 2 Immunosuppressive effect: including preventing or inhibiting cell-mediated immune responses, delayed allergic reactions, reducing T lymphocytes, monocytes, and eosinophils |
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