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Founded in:
2002-11-26 -
Country:
China -
Address:
No. 18, Yicheng Road, Chaohu City, Anhui Province -
Tax NO.:
91340181744869155N -
Registered Funds:
31 million yuan -
Website:
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Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| MOUTAN CORTEX |
It can counteract the ST segment elevation of the electrocardiogram after myocardial ischemia in rats induced by coronary artery ligation or intravenous injection of posterior pituitary hormone; prolong the survival time of mice in a normobaric hypoxic environment; and improve the auricle microcirculation disorder of mice induced by intravenous injection of high-molecular-weight dextran.
More
It can counteract the ST segment elevation of the electrocardiogram after myocardial ischemia in rats induced by coronary artery ligation or intravenous injection of posterior pituitary hormone; prolong the survival time of mice in a normobaric hypoxic environment; and improve the auricle microcirculation disorder of mice induced by intravenous injection of high-molecular-weight dextran. |
0 | ||
| Riligustilide |
It can counteract the ST segment elevation of the electrocardiogram after myocardial ischemia in rats induced by coronary artery ligation or intravenous injection of posterior pituitary hormone; prolong the survival time of mice in a normobaric hypoxic environment; and improve the auricle microcirculation disorder of mice induced by intravenous injection of high-molecular-weight dextran.
More
It can counteract the ST segment elevation of the electrocardiogram after myocardial ischemia in rats induced by coronary artery ligation or intravenous injection of posterior pituitary hormone; prolong the survival time of mice in a normobaric hypoxic environment; and improve the auricle microcirculation disorder of mice induced by intravenous injection of high-molecular-weight dextran. |
89354-45-0 | 0 | |
| Borneol |
It can counteract the ST segment elevation of the electrocardiogram after myocardial ischemia in rats induced by coronary artery ligation or intravenous injection of posterior pituitary hormone; prolong the survival time of mice in a normobaric hypoxic environment; and improve the auricle microcirculation disorder of mice induced by intravenous injection of high-molecular-weight dextran.
More
It can counteract the ST segment elevation of the electrocardiogram after myocardial ischemia in rats induced by coronary artery ligation or intravenous injection of posterior pituitary hormone; prolong the survival time of mice in a normobaric hypoxic environment; and improve the auricle microcirculation disorder of mice induced by intravenous injection of high-molecular-weight dextran. |
507-70-0 | 1 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Borneol |
Counteract post-ischemic electrocardiogram ST segment elevation; prolong the survival time of mice in a normobaric hypoxic environment and improve microcirculatory disorders.
More
Counteract post-ischemic electrocardiogram ST segment elevation; prolong the survival time of mice in a normobaric hypoxic environment and improve microcirculatory disorders. |
507-70-0 | 1 | |
| Riligustilide |
Counteract post-ischemic electrocardiogram ST segment elevation; prolong the survival time of mice in a normobaric hypoxic environment and improve microcirculatory disorders.
More
Counteract post-ischemic electrocardiogram ST segment elevation; prolong the survival time of mice in a normobaric hypoxic environment and improve microcirculatory disorders. |
89354-45-0 | 0 | |
| MOUTAN CORTEX |
Counteract post-ischemic electrocardiogram ST segment elevation; prolong the survival time of mice in a normobaric hypoxic environment and improve microcirculatory disorders.
More
Counteract post-ischemic electrocardiogram ST segment elevation; prolong the survival time of mice in a normobaric hypoxic environment and improve microcirculatory disorders. |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Clarithromycin |
This product is a macrolide antibiotic, which has inhibitory effects on Gram-positive bacteria such as Staphylococcus aureus, Streptococcus, and Pneumococcus, as well as some Gram-negative bacteria such as Haemophilus influenzae, Bordetella pertussis, Neisseria gonorrhoeae, Legionella pneumophila, and some anaerobic bacteria such as Bacteroides fragilis, Streptococcus peptostreptococcus, and Propionibacterium acnes. In addition, it also has inhibitory effects on mycoplasma. The characteristics of this product are that its antibacterial activity in vitro is similar to that of erythromycin, but its antibacterial activity in vivo against some bacteria such as Staphylococcus aureus, Streptococcus, and Haemophilus influenzae is stronger than that of erythromycin. There is cross-resistance between this product and erythromycin. The mechanism of action of this product is to inhibit the association of the 50S subunit of the nuclear protein and inhibit protein synthesis to produce an antibacterial effect.
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This product is a macrolide antibiotic, which has inhibitory effects on Gram-positive bacteria such as Staphylococcus aureus, Streptococcus, and Pneumococcus, as well as some Gram-negative bacteria such as Haemophilus influenzae, Bordetella pertussis, Neisseria gonorrhoeae, Legionella pneumophila, and some anaerobic bacteria such as Bacteroides fragilis, Streptococcus peptostreptococcus, and Propionibacterium acnes. In addition, it also has inhibitory effects on mycoplasma. The characteristics of this product are that its antibacterial activity in vitro is similar to that of erythromycin, but its antibacterial activity in vivo against some bacteria such as Staphylococcus aureus, Streptococcus, and Haemophilus influenzae is stronger than that of erythromycin. There is cross-resistance between this product and erythromycin. The mechanism of action of this product is to inhibit the association of the 50S subunit of the nuclear protein and inhibit protein synthesis to produce an antibacterial effect. |
81103-11-9 | 46 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Gentamicin sulfate |
It has antibacterial effects on various Gram-negative bacteria, Gram-positive bacteria and Helicobacter pylori. Its mechanism of action is to inhibit the synthesis of bacterial proteins.
More
It has antibacterial effects on various Gram-negative bacteria, Gram-positive bacteria and Helicobacter pylori. Its mechanism of action is to inhibit the synthesis of bacterial proteins. |
20mg | 1405-41-0 | 29 |
| Procaine hydrochloride |
It has a local anesthetic effect. Its mechanism of action is to competitively bind to the nerve membrane lipoprotein, block sodium ions, and hinder the conduction of nerve fibers.
More
It has a local anesthetic effect. Its mechanism of action is to competitively bind to the nerve membrane lipoprotein, block sodium ions, and hinder the conduction of nerve fibers. |
0.1g | 51-05-8 | 11 |
| Vitamin B12 |
It is a substance necessary for the formation of normal epithelial cells in the digestive tract
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It is a substance necessary for the formation of normal epithelial cells in the digestive tract |
0.02mg | 68-19-9 | 16 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Potassium chloride |
Potassium is the main cation in cells, with a concentration of 150-160 mmol/L; while the main cation outside cells is sodium ion, with a potassium concentration of only 3.5-5 mmol/L. The body mainly relies on Na+-K±ATPase on the cell membrane to maintain the concentration difference between K+ and Na+ in cells. The acid-base balance in the body has an impact on potassium metabolism. For example, when acidosis occurs, H+ enters the cell. In order to maintain the potential difference of the cell, K+ is released outside the cell, causing or aggravating hyperkalemia. Metabolic disorders can also affect acid-base balance. The normal intracellular and extracellular potassium ion concentration and concentration difference are closely related to some important functions of cells, including maintaining carbohydrate metabolism, glycogen storage, protein metabolism, intracellular osmotic pressure and acid-base balance, myocardial excitability and conductivity; maintaining normal skeletal muscle tension and nerve impulse conduction, and increasing the tension of intestinal, uterine and bronchial smooth muscles.
More
Potassium is the main cation in cells, with a concentration of 150-160 mmol/L; while the main cation outside cells is sodium ion, with a potassium concentration of only 3.5-5 mmol/L. The body mainly relies on Na+-K±ATPase on the cell membrane to maintain the concentration difference between K+ and Na+ in cells. The acid-base balance in the body has an impact on potassium metabolism. For example, when acidosis occurs, H+ enters the cell. In order to maintain the potential difference of the cell, K+ is released outside the cell, causing or aggravating hyperkalemia. Metabolic disorders can also affect acid-base balance. The normal intracellular and extracellular potassium ion concentration and concentration difference are closely related to some important functions of cells, including maintaining carbohydrate metabolism, glycogen storage, protein metabolism, intracellular osmotic pressure and acid-base balance, myocardial excitability and conductivity; maintaining normal skeletal muscle tension and nerve impulse conduction, and increasing the tension of intestinal, uterine and bronchial smooth muscles. |
7447-40-7 | 39 |