Fujian Nanshaolin Pharmaceutical Co., Ltd.
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Founded in:
1996-12-30 -
Country:
China -
Address:
No. 10, Fuzheng Road, Shizhu Street, Rongqiao Economic and Technological Development Zone, Fuqing City -
Tax NO.:
91350181705154309D -
Registered Funds:
100 million yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| DL-Methionine |
One of the eight essential amino acids for the human body, it combines with ATP to generate S-adenosine, promotes methylation of liver cell membrane phospholipids, reduces intrahepatic bile stasis, enhances transsulfurization, restores the physiological function of liver cells, promotes the disappearance of jaundice and recovery of liver function, supplies methyl to promote choline synthesis, promotes liver fat metabolism, protects the liver and detoxifies.
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One of the eight essential amino acids for the human body, it combines with ATP to generate S-adenosine, promotes methylation of liver cell membrane phospholipids, reduces intrahepatic bile stasis, enhances transsulfurization, restores the physiological function of liver cells, promotes the disappearance of jaundice and recovery of liver function, supplies methyl to promote choline synthesis, promotes liver fat metabolism, protects the liver and detoxifies. |
59-51-8 | 10 | |
| Thiamine chloride |
It combines with pyrophosphate in the body to form a cocarboxylase, participating in the oxidative decarboxylation reaction of pyruvate and α-ketoglutarate in glucose metabolism, and is necessary for glucose metabolism.
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It combines with pyrophosphate in the body to form a cocarboxylase, participating in the oxidative decarboxylation reaction of pyruvate and α-ketoglutarate in glucose metabolism, and is necessary for glucose metabolism. |
59-43-8 | 9 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Dexamethasone sodium phosphate |
Adrenal cortex hormone drugs. It has anti-inflammatory, anti-allergic, anti-rheumatic and immunosuppressive effects. Its mechanism of action is: 1. Anti-inflammatory effect: This product reduces and prevents the tissue's response to inflammation, thereby reducing the manifestations of inflammation. It can inhibit the accumulation of inflammatory cells, including macrophages and leukocytes, at the site of inflammation, and inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. 2. Immunosuppressive effect: including preventing or inhibiting cell-mediated immune responses, delayed allergic reactions, reducing the number of T lymphocytes, monocytes, and eosinophils, reducing the ability of immunoglobulins to bind to cell surface receptors, and inhibiting the synthesis and release of interleukins, thereby reducing the transformation of T lymphocytes into lymphoblasts.
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Adrenal cortex hormone drugs. It has anti-inflammatory, anti-allergic, anti-rheumatic and immunosuppressive effects. Its mechanism of action is: 1. Anti-inflammatory effect: This product reduces and prevents the tissue's response to inflammation, thereby reducing the manifestations of inflammation. It can inhibit the accumulation of inflammatory cells, including macrophages and leukocytes, at the site of inflammation, and inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. 2. Immunosuppressive effect: including preventing or inhibiting cell-mediated immune responses, delayed allergic reactions, reducing the number of T lymphocytes, monocytes, and eosinophils, reducing the ability of immunoglobulins to bind to cell surface receptors, and inhibiting the synthesis and release of interleukins, thereby reducing the transformation of T lymphocytes into lymphoblasts. |
55203-24-2 | 25 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Furosemide |
1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc. It mainly inhibits the active reabsorption of NaCl by the thick-walled segment of the renal tubular loop, reduces the osmotic pressure gradient difference of the medullary interstitial fluid, and leads to increased excretion of water, Na+, and Cl-; promotes increased Na+-K+ and Na+-H+ exchange in the distal tubule, which increases the excretion of K+ and H+; inhibits the reabsorption of Ca2+ and Mg2+ by the loop of Henle and increases their excretion. Short-term medication can increase uric acid excretion, and long-term medication can cause hyperuricemia. 2. Effect on hemodynamics: Furosemide can inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, and dilate blood vessels; dilate renal blood vessels, reduce renal vascular resistance, and increase renal blood flow; it does not reduce glomerular filtration rate while promoting diuresis; it can dilate pulmonary capacitance veins, reduce pulmonary capillary permeability, reduce the amount of blood returning to the heart, and reduce left ventricular end-diastolic pressure, which is helpful in the treatment of acute left heart failure and adult respiratory distress syndrome.
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1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc. It mainly inhibits the active reabsorption of NaCl by the thick-walled segment of the renal tubular loop, reduces the osmotic pressure gradient difference of the medullary interstitial fluid, and leads to increased excretion of water, Na+, and Cl-; promotes increased Na+-K+ and Na+-H+ exchange in the distal tubule, which increases the excretion of K+ and H+; inhibits the reabsorption of Ca2+ and Mg2+ by the loop of Henle and increases their excretion. Short-term medication can increase uric acid excretion, and long-term medication can cause hyperuricemia. 2. Effect on hemodynamics: Furosemide can inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, and dilate blood vessels; dilate renal blood vessels, reduce renal vascular resistance, and increase renal blood flow; it does not reduce glomerular filtration rate while promoting diuresis; it can dilate pulmonary capacitance veins, reduce pulmonary capillary permeability, reduce the amount of blood returning to the heart, and reduce left ventricular end-diastolic pressure, which is helpful in the treatment of acute left heart failure and adult respiratory distress syndrome. |
54-31-9 | 49 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Fluconazole |
This product is a new type of triazole antifungal drug that can specifically and effectively inhibit the synthesis of fungal sterols, that is, the synthesis of ergosterol, the main component of fungal cells. It also inhibits fungal peroxidase, leading to fungal death. Intravenous injection of this product is effective for various animal fungal infections, such as Candida infections (including systemic candidiasis and infections caused by immunosuppression in animals), Cryptococcus neoformans infections (including intracranial infections), Microsporum infections, and Trichophyton, Epidermophyton, Pityrosporum and other infections.
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This product is a new type of triazole antifungal drug that can specifically and effectively inhibit the synthesis of fungal sterols, that is, the synthesis of ergosterol, the main component of fungal cells. It also inhibits fungal peroxidase, leading to fungal death. Intravenous injection of this product is effective for various animal fungal infections, such as Candida infections (including systemic candidiasis and infections caused by immunosuppression in animals), Cryptococcus neoformans infections (including intracranial infections), Microsporum infections, and Trichophyton, Epidermophyton, Pityrosporum and other infections. |
86386-73-4 | 69 |