Histamine H2-receptor antagonists block the effect of histamine on parietal cell H2-receptors through a competitive inhibition mechanism, inhibiting basal gastric acid secretion during the day and at night as well as gastric acid secretion caused by food, histamine, pentagastrin, caffeine and insulin.

Sodium glycerophosphate is a phosphorus supplement used to meet the body's daily phosphorus needs. Phosphorus is involved in bone formation and in the composition of cell membranes in the form of phospholipids. Phosphorus is also related to the activity of many metabolic enzymes and plays a vital role in energy metabolism.

Caffeine can increase the content of cyclic adenosine monophosphate (cAMP) in cells. Small doses can enhance the excitation process of the cerebral cortex, invigorate the spirit, and relieve fatigue. Large doses can excite the medulla oblongata center.

Vitamin B1 combines with adenosine triphosphate to form vitamin B1 pyrophosphate, which is a necessary coenzyme for carbohydrate metabolism. Vitamin B1 can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, and the conduction of nerve impulses is impaired, affecting gastrointestinal and myocardial functions.

Nicotinic acid can be converted into nicotinamide in the body, and then combined with riboadenin to form nicotinamide adenine dinucleotide (coenzyme I) and nicotinamide adenine dinucleotide phosphate (coenzyme II), which are necessary for lipid, amino acid, protein, purine metabolism, tissue respiration oxidation and glycogenolysis. Nicotinic acid can reduce the effect of coenzyme A; it affects the transport of cholesterol in the blood by inhibiting the synthesis of very low density lipoprotein (VLDL). Nicotine also has the effect of peripheral vasodilation.

This product is an azole antifungal drug, which has antibacterial effects on Candida, Chromatiaceae, Coccidioides, Histoplasma, Sporothrix, etc., and also has antibacterial activity against Trichophyton, etc. It interferes with the activity of cytochrome P450, inhibits the biosynthesis of ergosterol, the main steroid of fungal cell membrane, damages the fungal cell membrane and changes its permeability, leading to leakage of important intracellular substances. It can inhibit the biosynthesis of triglycerides and phospholipids of fungi, inhibit the activity of oxidase and peroxidase, cause the accumulation of hydrogen peroxide in cells, and lead to cell submicrostructure degeneration and cell necrosis. For Candida albicans, it can inhibit the process of transformation from spores to invasive hyphae.