Iron is a component of hemoglobin in red blood cells. When iron is deficient, the amount of hemoglobin synthesized by red blood cells decreases, causing the red blood cells to become smaller and their oxygen-carrying capacity to decrease, resulting in iron deficiency anemia.

Participates in the formation of coenzymes in the body and has an auxiliary therapeutic effect on iron deficiency anemia

It can relax bronchial smooth muscles, shrink bronchial mucosal blood vessels, reduce congestion and edema, and improve airway obstruction.

It contains platycodon saponins, which can increase the secretion of respiratory mucus, dilute sputum, and make it easier to cough up.

It can reflexively increase the secretion of respiratory mucosal glands, making it easier to discharge sputum and facilitating the removal of sticky sputum.

Sodium glycerophosphate is a phosphorus supplement used to meet the body's daily phosphorus needs. Phosphorus is involved in bone formation and in the composition of cell membranes in the form of phospholipids. Phosphorus is also related to the activity of many metabolic enzymes and plays a vital role in energy metabolism.

The caffeine in this product can increase the intracellular cyclic adenosine monophosphate (cAMP) content. Small doses can enhance the excitement process of the cerebral cortex, invigorate the spirit, and relieve fatigue. Large doses can excite the medulla oblongata center.

Vitamin B1 combines with adenosine triphosphate to form vitamin B1 pyrophosphate, which is a necessary coenzyme for carbohydrate metabolism. Vitamin B1 can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, and the conduction of nerve impulses is impaired, affecting gastrointestinal and myocardial functions.

Nicotinic acid can be converted into nicotinamide in the body, and then combined with riboadenin to form nicotinamide adenine dinucleotide (coenzyme I) and nicotinamide adenine dinucleotide phosphate (coenzyme II), which are necessary for lipid, amino acid, protein, purine metabolism, tissue respiration oxidation and glycogenolysis. Nicotinic acid can reduce the effect of coenzyme A; it affects the transport of cholesterol in the blood by inhibiting the synthesis of very low density lipoprotein (VLDL). Nicotine also has the effect of peripheral vasodilation.

Increase the synthesis of GABA and reduce the degradation of GABA, thereby increasing the concentration of the inhibitory neurotransmitter g-aminobutyric acid (GABA), reducing the excitability of neurons and inhibiting seizures. It can produce an inhibitory effect on Na channels similar to phenytoin. It is harmful to the liver.