Antithyroid drugs. Its mechanism of action is to inhibit peroxidase in the thyroid gland, thereby preventing the iodination of tyrosine and the condensation of iodinated tyrosine in the thyroid gland, thereby inhibiting the synthesis of thyroxine. At the same time, it inhibits the conversion of T4 to T3 in peripheral tissues, causing the content of more active T3 in serum to decrease rapidly.

Antithyroid drugs. Its mechanism of action is to inhibit peroxidase in the thyroid gland, thereby preventing the iodination of tyrosine and the condensation of iodinated tyrosine in the thyroid gland, thereby inhibiting the synthesis of thyroxine. At the same time, it inhibits the conversion of T4 to T3 in peripheral tissues, causing the content of more active T3 in serum to decrease rapidly.

Iron is the main component of hemoglobin and myoglobin. It can accelerate hemoglobin synthesis and gradually correct symptoms related to tissue iron deficiency and decreased activity of iron-containing enzymes.

It can maintain and improve gastrointestinal function.

Sodium glycerophosphate is a phosphorus supplement used to meet the body's daily phosphorus needs. Phosphorus is involved in bone formation and in the composition of cell membranes in the form of phospholipids. Phosphorus is also related to the activity of many metabolic enzymes and plays a vital role in energy metabolism.

The caffeine in this product can increase the intracellular cyclic adenosine monophosphate (cAMP) content. Small doses can enhance the excitement process of the cerebral cortex, invigorate the spirit, and relieve fatigue. Large doses can excite the medulla oblongata center.

Vitamin B1 combines with adenosine triphosphate to form vitamin B1 pyrophosphate, which is a necessary coenzyme for carbohydrate metabolism. Vitamin B1 can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, and the conduction of nerve impulses is impaired, affecting gastrointestinal and myocardial functions.

Nicotinic acid can be converted into nicotinamide in the body, and then combined with riboadenin to form nicotinamide adenine dinucleotide (coenzyme I) and nicotinamide adenine dinucleotide phosphate (coenzyme II), which are essential for lipid, amino acid, protein, purine metabolism, tissue respiration oxidation and glycogenolysis. Nicotinic acid can reduce the effect of coenzyme A; it affects the transport of cholesterol in the blood by inhibiting the synthesis of very low density lipoprotein (VLDL). Nicotine also has the effect of peripheral vasodilation.