Calcium ion antagonists exert their pharmacological effects by regulating the influx of calcium ions on the cell membranes of myocardial conduction cells, myocardial contractile cells, and arterial smooth muscle cells, dilating coronary arteries, antagonizing coronary artery spasm, reducing total peripheral resistance, prolonging the effective refractory period of the atrioventricular node, reducing the ventricular rate of patients with chronic atrial fibrillation and atrial flutter, lowering blood pressure, reducing afterload, inhibiting myocardial contraction, and improving left ventricular diastolic function.

It promotes the biochemical synthesis of hexosamine and glucosamine, the components of gastrointestinal mucosal epithelium, and has a protective and repairing effect on gastrointestinal mucosal damage. In the pylorus ligated rat experiment, it can inhibit the ulcers caused by aspirin and indomethacin. This compound preparation has the effect of promoting the healing of ulcers in various experimental rat ulcers, indicating that it has a therapeutic effect on low-acid (acid-free) ulcers. In addition, this compound preparation can inhibit the production of pepsinogen in the gastric mucosa of rats.

It can directly act on the inflamed mucosa and has an anti-inflammatory effect. Animal experiments show that it can effectively inhibit ulcers caused by aspirin. This compound preparation has the effect of promoting the healing of ulcers in various experimental rat ulcers, indicating that it has a therapeutic effect on low-acid (acid-free) ulcers. In addition, this compound preparation can inhibit the production of pepsinogen in the gastric mucosa of rats.

Neutralizes stomach acid

Neutralizes stomach acid

Promote gastric mucosal regeneration

Antispasmodic and analgesic

Antispasmodic and analgesic

Protect gastric mucosa, astringent and hemostatic

Aids digestion

Aids digestion

Relieve stomach discomfort

This product is a drug that inhibits the synthesis of uric acid. Allopurinol and its metabolite oxypurinol can inhibit xanthine oxidase, preventing hypoxanthine and xanthine from being metabolized into uric acid, thereby reducing the production of uric acid. It reduces the uric acid content in the blood and urine to a level below the solubility, prevents uric acid from forming crystals and depositing in joints and other tissues, and also helps to redissolve uric acid crystals in the tissues of gout patients. Allopurinol also inhibits the synthesis of new purines in the body by acting on hypoxanthine-guanine phosphate nucleic acid convertase.