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Founded in:
2006-12-22 -
Country:
China -
Address:
No. 1739, Middle Section of Kunlunshan Avenue, Lanzhou New District, Lanzhou City, Gansu Province -
Tax NO.:
916200007948829218 -
Registered Funds:
84.5 million yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Angelicae Sinensis Radix |
|
0 | ||
| Sucrose |
|
57-50-1 | 73 | |
| Starch |
|
9005-25-8 | 79 | |
| Dextrin |
|
9004-53-9 | 50 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Dexamethasone sodium phosphate |
Adrenal cortex hormone drugs. It has anti-inflammatory, anti-allergic, anti-rheumatic and immunosuppressive effects. 1. Anti-inflammatory effect: This product reduces and prevents tissue response to inflammation, thereby reducing the manifestations of inflammation. It can inhibit the accumulation of inflammatory cells, including macrophages and leukocytes, at the site of inflammation, and inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. 2. Immunosuppressive effect: including preventing or inhibiting cell-mediated immune responses, delayed allergic reactions, reducing the number of T lymphocytes, monocytes, and eosinophils, reducing the ability of immunoglobulins to bind to cell surface receptors, and inhibiting the synthesis and release of interleukins, thereby reducing the transformation of T lymphocytes into lymphoblasts.
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Adrenal cortex hormone drugs. It has anti-inflammatory, anti-allergic, anti-rheumatic and immunosuppressive effects. 1. Anti-inflammatory effect: This product reduces and prevents tissue response to inflammation, thereby reducing the manifestations of inflammation. It can inhibit the accumulation of inflammatory cells, including macrophages and leukocytes, at the site of inflammation, and inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. 2. Immunosuppressive effect: including preventing or inhibiting cell-mediated immune responses, delayed allergic reactions, reducing the number of T lymphocytes, monocytes, and eosinophils, reducing the ability of immunoglobulins to bind to cell surface receptors, and inhibiting the synthesis and release of interleukins, thereby reducing the transformation of T lymphocytes into lymphoblasts. |
55203-24-2 | 25 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Midecamycin A1 |
The antibacterial property is similar to that of erythromycin. It has a strong antibacterial effect on Gram-positive bacteria, and is effective against Staphylococcus, Streptococcus pyogenes, Streptococcus viridans, Streptococcus pneumoniae, Diphtheria bacillus, Tetanus bacillus, and anthrax bacillus. It also has antibacterial effects on Gram-negative bacteria such as gonococci and pertussis bacillus. It is also effective against Leptospira, Rickettsia, and Mycoplasma. It can be used as a substitute for erythromycin for infections of the oropharynx, respiratory tract, skin and soft tissue, and bile duct caused by the above-mentioned sensitive bacteria.
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The antibacterial property is similar to that of erythromycin. It has a strong antibacterial effect on Gram-positive bacteria, and is effective against Staphylococcus, Streptococcus pyogenes, Streptococcus viridans, Streptococcus pneumoniae, Diphtheria bacillus, Tetanus bacillus, and anthrax bacillus. It also has antibacterial effects on Gram-negative bacteria such as gonococci and pertussis bacillus. It is also effective against Leptospira, Rickettsia, and Mycoplasma. It can be used as a substitute for erythromycin for infections of the oropharynx, respiratory tract, skin and soft tissue, and bile duct caused by the above-mentioned sensitive bacteria. |
0 | ||
| Sineptina A6 |
The antibacterial property is similar to that of erythromycin. It has a strong antibacterial effect on Gram-positive bacteria, and is effective against Staphylococcus, Streptococcus pyogenes, Streptococcus viridans, Streptococcus pneumoniae, Diphtheria bacillus, Tetanus bacillus, and anthrax bacillus. It also has antibacterial effects on Gram-negative bacteria such as gonococci and pertussis bacillus. It is also effective against Leptospira, Rickettsia, and Mycoplasma. It can be used as a substitute for erythromycin for infections of the oropharynx, respiratory tract, skin and soft tissue, and bile duct caused by the above-mentioned sensitive bacteria.
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The antibacterial property is similar to that of erythromycin. It has a strong antibacterial effect on Gram-positive bacteria, and is effective against Staphylococcus, Streptococcus pyogenes, Streptococcus viridans, Streptococcus pneumoniae, Diphtheria bacillus, Tetanus bacillus, and anthrax bacillus. It also has antibacterial effects on Gram-negative bacteria such as gonococci and pertussis bacillus. It is also effective against Leptospira, Rickettsia, and Mycoplasma. It can be used as a substitute for erythromycin for infections of the oropharynx, respiratory tract, skin and soft tissue, and bile duct caused by the above-mentioned sensitive bacteria. |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Furosemide |
1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chlorine, potassium, calcium, magnesium, phosphorus, etc.; 2. Effect on hemodynamics: It has a vasodilator effect, can inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, thereby dilating renal blood vessels, reducing renal vascular resistance, and increasing renal blood flow. In addition, it can reduce the permeability of pulmonary capillaries, reduce the amount of blood returning to the heart, and reduce the end-diastolic pressure of the left ventricle, which is helpful for the treatment of acute left heart failure and adult respiratory distress syndrome.
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1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chlorine, potassium, calcium, magnesium, phosphorus, etc.; 2. Effect on hemodynamics: It has a vasodilator effect, can inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, thereby dilating renal blood vessels, reducing renal vascular resistance, and increasing renal blood flow. In addition, it can reduce the permeability of pulmonary capillaries, reduce the amount of blood returning to the heart, and reduce the end-diastolic pressure of the left ventricle, which is helpful for the treatment of acute left heart failure and adult respiratory distress syndrome. |
54-31-9 | 49 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Acetaminophen |
It inhibits the synthesis and release of prostaglandins (PGE2) in the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating to achieve an antipyretic effect. It can also inhibit the effects of bradykinin, increase the pain threshold and produce analgesic effects.
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It inhibits the synthesis and release of prostaglandins (PGE2) in the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating to achieve an antipyretic effect. It can also inhibit the effects of bradykinin, increase the pain threshold and produce analgesic effects. |
63mg | 103-90-2 | 68 |
| Acetylsalicylic acid |
The mechanism of action of acetaminophen in this product is to inhibit the synthesis and release of prostaglandins (PGE2) in the hypothalamic temperature regulation center, thereby producing peripheral vasodilation and inducing sweating to achieve an antipyretic effect. At the same time, it can inhibit the effects of bradykinin, etc., increase the pain threshold and produce analgesic effects.
More
The mechanism of action of acetaminophen in this product is to inhibit the synthesis and release of prostaglandins (PGE2) in the hypothalamic temperature regulation center, thereby producing peripheral vasodilation and inducing sweating to achieve an antipyretic effect. At the same time, it can inhibit the effects of bradykinin, etc., increase the pain threshold and produce analgesic effects. |
115mg | 50-78-2 | 35 |
| Caffeine |
The mechanism of action of acetaminophen in this product is to inhibit the synthesis and release of prostaglandins (PGE2) in the hypothalamic temperature regulation center, thereby producing peripheral vasodilation and inducing sweating to achieve an antipyretic effect. At the same time, it can inhibit the effects of bradykinin, etc., increase the pain threshold and produce analgesic effects.
More
The mechanism of action of acetaminophen in this product is to inhibit the synthesis and release of prostaglandins (PGE2) in the hypothalamic temperature regulation center, thereby producing peripheral vasodilation and inducing sweating to achieve an antipyretic effect. At the same time, it can inhibit the effects of bradykinin, etc., increase the pain threshold and produce analgesic effects. |
15mg | 0 |