It inhibits the synthesis and release of prostaglandins (PGE2) in the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating to achieve an antipyretic effect. It can also inhibit the effects of bradykinin, increase the pain threshold and produce analgesic effects.

The mechanism of action of acetaminophen in this product is to inhibit the synthesis and release of prostaglandins (PGE2) in the hypothalamic temperature regulation center, thereby producing peripheral vasodilation and inducing sweating to achieve an antipyretic effect. At the same time, it can inhibit the effects of bradykinin, etc., increase the pain threshold and produce analgesic effects.

The mechanism of action of acetaminophen in this product is to inhibit the synthesis and release of prostaglandins (PGE2) in the hypothalamic temperature regulation center, thereby producing peripheral vasodilation and inducing sweating to achieve an antipyretic effect. At the same time, it can inhibit the effects of bradykinin, etc., increase the pain threshold and produce analgesic effects.

It achieves bactericidal effect by inhibiting biosynthesis of sensitive bacterial cell walls. It has obvious synergistic effect when used in combination with sulbactam, which can enhance the antibacterial effect against certain drug-resistant strains.

It has an irreversible inhibitory effect on most important β-lactamases produced by β-lactam antibiotic-resistant strains, protecting β-lactam antibiotics from hydrolysis. It can also bind to certain penicillin-binding proteins to enhance the effect of compound preparations on sensitive strains.

It is a third-generation cephalosporin that achieves bactericidal effect by inhibiting the biosynthesis of sensitive bacterial cell walls. When used in combination with sulbactam, it has a significant synergistic effect and enhances the effect on drug-resistant strains.

It has an irreversible inhibitory effect on most important β-lactamases produced by β-lactam antibiotic-resistant strains, protecting β-lactam antibiotics from hydrolysis and destruction by β-lactamases of resistant bacteria. It can be combined with certain penicillin-binding proteins to enhance the antibacterial effect on sensitive strains.

Bactericidal effect achieved by inhibiting the biosynthesis of sensitive bacterial cell walls

It has an irreversible inhibitory effect on most important β-lactamases produced by β-lactam antibiotic-resistant strains, protecting β-lactam antibiotics from hydrolysis and destruction by β-lactamases of resistant bacteria; it can combine with certain penicillin-binding proteins to enhance the sensitivity of this compound preparation

It achieves bactericidal effect by inhibiting biosynthesis of sensitive bacterial cell walls. It has synergistic effect when used in combination with sulbactam, enhancing the antibacterial effect.

It has an irreversible inhibitory effect on most important β-lactamases produced by β-lactam antibiotic-resistant strains, protects β-lactam antibiotics from hydrolysis and enhances antibacterial activity. It can also combine with certain penicillin-binding proteins to enhance the effect of compound preparations on sensitive strains.

This product has the effects of inhibiting Ca influx and anti-sympathetic nerves. It can reduce myocardial contractility and relax vascular smooth muscle, increase coronary flow, and reduce myocardial oxygen consumption. It also has the effect of promoting collateral circulation and can be used for the prevention and treatment of angina pectoris.

Adrenal cortex hormone drugs. It has anti-inflammatory, anti-allergic, anti-rheumatic and immunosuppressive effects. Its mechanism of action is as follows: 1. Anti-inflammatory effect: This product reduces and prevents tissue response to inflammation, thereby reducing the manifestations of inflammation. It can inhibit the accumulation of inflammatory cells, including macrophages and leukocytes, at the site of inflammation, and inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. 2. Immunosuppressive effect: including preventing or inhibiting cell-mediated immune responses, delayed allergic reactions, reducing the number of T lymphocytes, monocytes, and eosinophils, reducing the ability of immunoglobulins to bind to cell surface receptors, and inhibiting the synthesis and release of interleukins, thereby reducing the transformation of T lymphocytes into lymphoblasts.