The ammonium ferric citrate contained in this product is an iron supplement. Iron is a component of hemoglobin in red blood cells. When iron is deficient, the amount of hemoglobin synthesized by red blood cells decreases, causing the red blood cells to become smaller and their oxygen-carrying capacity to decrease, resulting in iron deficiency anemia.

Vitamin B1 is involved in the formation of coenzymes in the body and has an auxiliary therapeutic effect on iron deficiency anemia.

It can lubricate and stimulate the intestinal wall, soften the stool and make it easier to pass.

It can lubricate and stimulate the intestinal wall, soften the stool and make it easier to pass.

Vitamin B1 combines with adenosine triphosphate to form vitamin B1 pyrophosphate, which is a necessary coenzyme for carbohydrate metabolism. Vitamin B1 can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, and the conduction of nerve impulses is impaired, affecting gastrointestinal and myocardial functions.

Sodium glycerophosphate is a phosphorus supplement used to meet the body's daily phosphorus needs. Phosphorus is involved in bone formation and in the composition of cell membranes in the form of phospholipids. Phosphorus is also related to the activity of many metabolic enzymes and plays a vital role in energy metabolism.

Caffeine can increase the content of cyclic adenosine monophosphate (cAMP) in cells. Small doses can enhance the excitation process of the cerebral cortex, invigorate the spirit, and relieve fatigue. Large doses can excite the medulla oblongata center.

Vitamin B1 combines with adenosine triphosphate to form vitamin B1 pyrophosphate, which is a necessary coenzyme for carbohydrate metabolism. Vitamin B1 can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, and the conduction of nerve impulses is impaired, affecting gastrointestinal and myocardial functions.

Nicotinic acid can be converted into nicotinamide in the body, and then combined with riboadenin to form nicotinamide adenine dinucleotide (coenzyme I) and nicotinamide adenine dinucleotide phosphate (coenzyme II), which are essential for lipid, amino acid, protein, purine metabolism, tissue respiration oxidation and glycogenolysis. Nicotinic acid can reduce the effect of coenzyme A; it affects the transport of cholesterol in the blood by inhibiting the synthesis of very low density lipoprotein (VLDL). Nicotine also has the effect of peripheral vasodilation.