Anti-hormone drugs. The mechanism of ovulation stimulation of this product is not fully understood. Since this product has a weak excitatory and strong antagonistic effect on estrogen, ovulation stimulation may be in the hypothalamus. First, antagonism is dominant. By competitively occupying the hypothalamic estrogen receptors, it interferes with the negative feedback of endogenous estrogen, promotes the secretion of luteinizing hormone and follicle-stimulating hormone, and then stimulates the growth of follicles. After the follicles mature, the release of estrogen increases, and the release of preovulatory gonadotropin is stimulated by positive feedback to reach a peak, thus ovulation. The treatment of male infertility may be related to the increase of FSH and LH and the promotion of sperm production.

It has a similar chemical structure to aldosterone, competitively inhibits aldosterone in the distal convoluted tubule and collecting duct, thereby interfering with the effect of aldosterone on promoting sodium reabsorption in the above-mentioned parts. It promotes the excretion of Na+ and Cl- to produce diuresis. Because the Na+K+ exchange mechanism is inhibited, the excretion of potassium is reduced, so it is a potassium-retaining diuretic. This product is a low-efficiency diuretic and has a therapeutic effect on edema caused by secondary aldosterone elevation.

Extract from the above information

No specific pharmacological effects mentioned

Artificially synthesized androgens can promote the development and maturation of male organs and secondary sexual characteristics; counteract estrogen, inhibit endometrial growth and pituitary gonadal function; promote protein synthesis and bone formation; stimulate bone marrow hematopoiesis, increase red blood cells and hemoglobin. The ratio of androgens to protein assimilation is 1:1.